With the stroke of a pen, President Obama could stop the needless “HIV” deaths, and save hundreds of billions.

Andrew Maniotis, Ph.D.

Associate Research Professor of Bioengineering;

Program of tumor mechanics and tissue regeneration;

234 SEO, MC 063

University of Illinois at Chicago, Chicago, IL 60607

Secure Email: andy.maniotis@gmail.com

University Email: amanioti@uic.edu

Cell: 773-960-9084

With the removal and resignation of Mark Dybul, the architect and executioner of George Bush’s PEPFAR initiative, I would like to offer my willingness to take up the task of his position in the new Obama administration, and provide some analysis of several very expensive and wasteful programs that currently are in place, whose monies could be redirected to generate far more effective programs of health and humanitarian aide.

It should be stated from the start that I would organize the program on the basis of suggestions promoted by organizations like Doctors Without Borders, rather than continue Global AIDS initiatives, as they have been directed for years, according to questionable science, and pharmaceutical interests.

It also should be stated first, that I recognize and acknowledge that since the emergence of AIDS, many of the assumptions and efforts that have been advanced to address the issue of “HIV/AIDS” have revealed genuine concern for “the AIDS people,” and others of the world, regardless of how misguided and destructive some of these assumptions and efforts have been.

Despite AIDS control efforts that have been based upon faith-based assumptions instead of solid scientific evidence, it is clear that such public health initiatives are borne from altruistic impulses, and mostly administered by persons who want to help, rather than to exploit or profit.

Having acknowledged that we are only human, one may begin this task of overhauling World AIDS initiatives by asking President Obama’s new science advisors if they intend to maintain the violation of human rights that is caused by “HIV” molecular testing of so-called “low-risk” and “high-risk” individuals. “HIV” test kits cross-react non-specifically with other factors and known disease syndromes.

Among “HIV” test subjects, with respect to the accuracy of diagnosis, complete disclosure to human test subjects should be made about the facts that:

1. There are at least 80+ known false positive “HIV” cross-reactions (please see Christine Johnson’s list of reasons to test false positive at the end of this document);

2. To obtain an unequivocally positive “HIV” test result each of the following potential chemical reactions should be eliminated through differential diagnosis as a possible reason for a false-positive test result on the ELISA (Enzyme Linked Immunoabsorbant Assay, WB’s (WESTERN BLOT), or PCR-based tests (Polymerase Chain Reaction), before a positive “HIV” diagnosis is made:

the spurious detection of p18, p24, p55, p12, p32, p51, p66 and gp160, gp41, gp120 antigens (antigens are molecules) that also may be present in fluids obtained from patients who are pregnant, from patients who suffer from other acute viral infections, from patients who have had recent flu or hepatitis B vaccinations, or for no reason that is associated with disease.

In 1993, U.S health authorities expanded AIDS diagnoses to include a category of people having but two laboratory conditions: a low white blood cell count (low WBC) and a test showing “HIV” antibodies of a high concentration. Thus these two test results, graded and judged on an arbitrary scale, despite a person having excellent clinical health and with no symptoms of disease whatsoever, stamp one in the U.S. to have “AIDS.” Then, despite beginning in fine health, the nightmare of toxic drug therapies is initiated, with scared witless new patients conjuring up ludicrous dim memories of perhaps a brief sexual fling, blood transfusion, or bloody injury often decades ago. By year 2000, this singular low WBC rogue category of AIDS (the last year this data was available) cited by

New York City Department of Health data was 90% of their AIDS cases, and rising.

This World health disaster called the AIDS era, and agony on a personal level, continues every day, brought by anti-retroviral drugs and mindless vaccine trials that should be instantly stopped or minimized. U.S. AIDS cases and deaths continue to be needlessly created by the 1993 AIDS definition which in a single day in 1993 caused a 204 percent increase in AIDS cases reported since the implementation of the new federal AIDS surveillance definition

(AIDS Alert (06/93) Vol. 8, No. 6, P. 81). This “Challenger-sized disaster” continues with the current useless tests, with Congress and top health officials conducting business as usual, and blind to victory over AIDS throughout Europe, Australia, and even right next door in Canada, because the U.S. model of AIDS is in error. The new President Barack Obama must demand complete reassessment of this monumental medical fiasco, and stop the mindless massacre, racism, and homophobia that is reinforced by “high-risk” and “low-risk” designations, and the tragically flawed American AIDS model.

For starters, any new Global AIDS coordinator working for the Obama administration should immediately demand a reappraisal of:

A.The failure to evoke seroconversion, in most cases T-cell activation, or protection, even after sixty-three multi-million dollar vaccine failures all have raised issue with the American AIDS model and the “HIV=AIDS” hypothesis (see 1995 Congress of the United States: Office of Technology assessment. Adverse Reactions to HIV Vaccines: Medical, Ethical, and Legal Issues. Roger C. Herdman, Director; and also the list of 33 current, mostly aborted, vaccine trials on the NIAD website.

B.The failure to isolate “HIV” from all other objects in the Universe, or to explain what the confusing presence of “HIV” antigens or nucleic acids in healthy drug-naïve persons means, or explain why normal cellular proteins like actin are found inside “HIV” particles (see Gluschankof P, Mondor I, Gelderblom HR, Sattentau QJ. Cell membrane vesicles are a major contaminant of gradient-enriched human immunodeficiency virus type-1 preparations. Virology. Mar 31;230(1):125-33, 1997; or Bess JW Jr, Gorelick RJ, Bosche WJ, Henderson LE, Arthur LO. Microvesicles are a source of contaminating cellular proteins found in purified HIV-1 preparations. Virology, Mar 31; 230(1):134-44, 1997);

C.The failure to appreciate, that the association of a molecular marker with any disease state, does not prove, disprove, or even suggest causality;

D.The failure of the 2008 Nobel committee to appreciate, in the case of awarding the recent Nobel Prize to Luc Montagnier and Barre-Sinoussi, that according to their 1983 paper, their Patient One’s “viral” isolate,” was derived from a fellow with swollen lymph nodes, a history of syphilis and syphilis treatment the year before, a history of gonorrhea, a history of cytomegalovirus infection, a history of herpes I and II infection, a history of Epstein-Barr virus infection, and God knows what else (Barre-Sinoussi, F,J.C. Chermann J.C., Rey F., Nugeyre MT., Chamaret, S., Gruest J., Dauguet C., Axler-Blin C., Vezinet-Brn F., Rouzioux, C., Rozenbaum R., & Montagnier L. Isolation of a T-lymphotropic retrovirus from a patient at risk for Acquired Immune Deficiency Syndrome. Science 230: 868-871, 1983);

E. The failure of the December 2008 Nobel committee to appreciate that by their granting of either the first half of their Nobel Prize award to Montagnier, Barre-Sinoussi, and second half to Harald Zur Hausen, that despite the mistaken link between “HIV” and six different cancers, or HPV with cervical cancer, that “HIV” has never been linked to one of the first two “AIDS-defining illnesses” (Kaposi’s sarcoma), and that “HPV” has never been shown to transform cells in a scientifically acceptable experiment, and that the reason why “HPV” (human papillomavirus) molecular sequences are sometimes associated with cervical cancers is because these are all simply endogenous molecular sequences kicked out by transformed cells, not Human immune deficiency or transforming papillomaviruses. In this context, HPV “virus” particles, to date have not been shown to induce cervical cells or any other kind of cells to become cancerous in an acceptable experiment no more than “HIV” has even been shown to transform any human cells. Also in this context, the Nobel committee should be held accountable about the fact that many of us in the scientific community know that it is simply mindless greed and conflicting interests that has appeared to motivate this last December’s Nobel Committee NOT to acknowledge and heed the widely publicized warnings on all of the more than 30 “HIV” package inserts that claim their kits cannot detect “HIV,” and ignore such agencies as The “College of American Pathologists (CAP),” and senior investigators at the National Cancer Institute (Dr. Mark Shiffman), Dr. Mark Stoler at the Virginia Healthcare system, and Attila Lorincz, PhD, chief scientific officer and senior VP of research development at Digene the company which makes “HPV” molecular test kits, that neither “HIV -sequences” or “HPV-sequences” have been validated against the clinical occurrence of AIDS or clinical cervical cancer (several of the Nobel committee had direct ties to AstraZeneca and other biopharmaceutical companies and “HIV” vaccine trails). In this context, the Nobel committee also failed to appreciate the shameful carnage currently being perpetrated by the so-called first “anti-cancer” vaccine GARDASIL (made by the same company Merck, who 20 years ago claimed that their hepatitis B vaccine was the first “anti-cancer” vaccine, before 15,000 French citizens filed a class action suit to their Health Minister to stop the hepatitis B vaccine mandate for its youngest citizens, because it harmed so many. In this context, it also might also be mentioned that the hepatitis B “first anti-cancer” vaccine topped the CDC’s list as being linked to the most reported vaccine adverse events after the 1999’s impassioned crusade to vaccine infants against “STD’s” or drug and alcohol-acquired syndromes like hepatitis B, and currently, Gardasil, Merck’s other “first anti-cancer” vaccine, which will be bought for the Nations poorest girls (read African Americans predominantly) at a public-funded price of at least 2 billion, already has been responsible for 78 outbreaks of genital warts (http://www.NaturalNews.com/024774.html” target=”_blank” title=”)

and, according to Judicial Watch:

“In June, 2008, the reported (<10% of adverse reactions are known to be reported) adverse events tally attributed to GARDASIL increased to 8,864: (http://tinyurl.com/6dkht7)”


Death toll linked to Gardasil vaccine rises. Complications include shock, ‘foaming at mouth,’ convulsions, coma.

Posted: June 30, 2008 10:18 pm Eastern, 2008 WorldNetDaily.

“Anaphylactic shock,” “foaming at mouth,” “grand mal convulsion,” “coma” and “now paralyzed” are a few of the startling descriptions included in a new federal report describing the complications from Merck & Co.’s Gardasil medication for sexually transmitted human papillomavirus – which has been proposed as mandatory for all schoolgirls.

The document was obtained from the U.S. Food and Drug Administration by Judicial Watch, a Washington group that investigates and prosecutes government corruption, and it has details of 10 deaths just since September.

“Given all the questions about Gardasil, the best public health policy would be to re-evaluate its safety and to prohibit its distribution to minors. In the least, governments should rethink any efforts to mandate or promote this vaccine for children,” said Judicial Watch President Tom Fitton.

The organization’s work uncovered reports of about one death each month since last fall, bringing the total death toll from the drug to at least 18 and as many as 20. There also were 140 “serious” reports of complications including about three dozen classified as life- threatening, 10 spontaneous abortions and half a dozen cases of Guillain-Barre Syndrome.

The document reveals the case of an 18-year-old woman who got the Gardasil vaccine, was found unconscious that evening, and died. Another woman, age 19, got the drug and the next morning was found dead in her bed.

The new documents also reveal a total of 8,864 Vaccine Adverse Event Reporting System records, up from a total of 3,461 that had been reported in a document just last fall.

F. The failure to sequence the “HIV” genome as a consistent pattern or genomic sequence, or to identify specific proteins that are not also found in normal, “non-infected” contexts. For instance,The DIADS culturing manual for “HIV” infection states that a true test reading is obtained when:

Two consecutive HIV p24 antigen VQA CORRECTED values of > 30 pg/ml (from a healthy donor source), of which the second value is at least four times greater than the first value or out of range” (O.D.>2) or

Two consecutive HIV p24 antigen VQA CORRECTED values (from a healthy donor source) that are out of range” (Optical density > 2); or

Three consecutive HIV p24 antigen VQA CORRECTED values of > 30 pg/ml (from a healthy donor source), where neither consecutive value is >than four times the previous sample, but the third value is at least four times greater than the first.

Thus, under current rules, if someone should have sex without informing the other participant(s) of their more than 30pg/ml p24 status, the “HIV-positive” person can be arrested by the State, thrown in jail, and tried and convicted for murder, if pregnant, a woman may be urged or even forced to abort, or to have a hysterectomy, as in the Serrano case. If less than 30pg/ml p24 (“HIV-capsid protein”) is detected, one can donate blood or sleep with everybody one can without telling them their less than 30 status, which constitutes p24 protein discrimination, rather than a positive “HIV” test result indicating the presence of an exogenous retroviral infection;

G.The failure to inform the public (and most scientists) that reverse transcriptase is not specific to viruses, nor are the gag, pol, env, p24, and other so-called “HIV-specific” genes and their products, which all can be detected in normal, “non- infected” contexts, and which are published on Medline;

H.The failure to block transmission of “HIV” or AIDS in mother to child transmission studies (MTCT) as shown by the Cochran Meta-analysis and other peer-reviewed reports, which almost without exception showed increased “HIV mutation rates” after black box label drugs such as nevirapine were discontinued in the U.S. because of their toxicitiy, and then ashamedly were administered to more than eight hundred seventy five thousand African mother-infant pairs by Max Essex of Harvard, and others.

I. The failure to acknowledge, appreciate, or investigate that safety officers of the NIH, such as Dr. Fishbein, who monitored the nevirapine trials as a safety officer, were fired, while those individuals such as Edmond Tremont who directed the nevaripine trial(s) were not even reprimanded after he had changed the data in safety reports that Dr. Fishbein and others had uncovered, in order to push forward George Bush’s and Mark Dybul’s PEPFAR pogrom and their abstinence-biblical-practice-enriched eugenics pogrom on Africans;

J. The failure to understand why ARV’s (anti-retrovirals) in some individuals, can prevent “AIDS syndromes,” because of their toxicity to normal immune cells can block these cells from expressing “HIV-specific” molecules as a normal response to a physiological or pharmaceutical stress on lymphocytes, or are detected as evidence of a rare genetic polymorphism, and the failure to appreciate that as shown in the Fischl, Veterans Affairs, Concorde, and first decade of HAART, that these drugs are so toxic, that they can in some individuals suppress both fungal and bacterial growth, but cannot prevent theoretical virus proliferation, because if the “HIV” paradigm is correct, these genomes of “HIV” would be rapidly integrated into the DNA of “infected” cells and are “out of reach” of drugs designed against “HIV” “molecules;”

K. The failure of John Moore’s “Hail Mary-discovered” microbicides, condom campaigns, and circumcision, to reduce “HIV-transmission” that have more often than not, increased the rate of detecting “HIV’s” molecular markers, instead of decreasing them among African human “lab rats;”

L.The failure to appreciate the disaster and infant mortality caused by breast feeding dissuasion campaigns, designed to decrease infant mortality from “HIV-infection,” but which increased infant mortality 20 times in formula fed infants, compared to mother-infant pairs that didn’t listen to their doctors, and who weren’t dissuaded from breast feeding, and the failure to appreciate the corresponding terrorism that has been waged against new mothers to promote formula dumping on 3rd World nations;

M.The failure to acknowledge how projected and WHO-manufactured “HIV” andAIDS” prevalence and incidence rates have not materialized, and how they recently have been dismissed by world AIDS leaders such as Kevin de Cock as signaling the end of the “heterosexual AIDS era” (except of course among people of African dissent or homosexuals who have been selectively biased during “HIV” testing campaigns, or selectively targeted duringHIV-preventative” microbicide or circumcision campaigns, or manufactured from “best guess estimates” based on STD clinics or perinatal clinics);

N. The failure to explain how “HIV’s” long “slow-virus-like” latency makes sense from any biochemical point of view;

O. The failure to support the progress of Doctors Without Borders, who recently showed how the cheap food supplement plumpynut, when given to the children of Niger, the poorest nation in Africa, has reversed the infant mortality rate, and without antibiotics or drugs, or without significant funding, as was revealed on 60 minutes;

P. The failure to develop a consistent In Vitro model to detect “HIV” infection;

Q. The failure to develop any “HIV” animal model, while “HIV” exposed chimps now rest in their 27 million dollar retirement homes because they never developed AIDS after injection 20 years ago with AIDS patient sera or “HIV;”

R. The failure of the biomedical establishment to offer and provide support to pursue and fund at least 17 other hypotheses that have explained or have even reversed in some cases, Acquired Immune Deficiency Syndrome;

S. The failure to pursue and fund rational and inexpensive patient-specific treatment regimens such as those developed in the German drug-abuse clinics by Heinrich Kremer, Juliane Sacher, or in African in Niger, by Doctors Without Borders who fed starving children plumpynet, and by many others who have shown they can reverse immunosuppression non-toxically, and with a minimum, or in most cases, with a complete lack of HAART;

T. The failure to appreciate why prostitutes and sex workers don’t acquireHIV’s” molecular markers, or develop “AIDS,” unless they are also chronic immunosuppressive illicit or pharmaceutical drug users or abusers;

U. The failure to account for why Human “HIV” transmission studies like the 10 year Padian study and many others have not shown “HIV” or “AIDS” transmission between serodiscordant couples, or among health care workers inoculated with “HIV-tainted” blood, or why the spouses of “HIV-positive” hemophiliacs and “HIV-negative” partners have failed to seroconvert or develop AIDS after numerous unprotected and repeated exposures to their “HIV” positive spouses;

V. The failure to address the phenomenon announced as recently as February 14th, 2008, in San Diego, California, when the local county health department made quite a big deal out of the fact that all sexually transmitted diseases in their local gay community have risen by an astounding 800 percent since 2003, including syphilis, gonorrhea, and chlamydia, while “HIV” infection rates have dropped since 2003 in the very same gay community;

W. The failure to explain how there can be large numbers of so-called “Long-Term-Non-Progressors,” or “Elite Controllers,” who never acquire any illness, although they may test positive for “HIV’s” molecular signature for more than two decades, or how it is possible that ICL-AIDS patients test negative for “HIV” but are thought to have “AIDS;”

X. The failure to account for how T-cell numbers or “viral load” don’t indicate any effect of a viral presence or infection, or explain why viral load continues to be aggressively monitored despite the fact that no virus has ever been observed in the blood of a so-called “HIV-positive” individual harboring high “viral load” as measured by PCR (polymerase chain reaction);

Y.The failure of the AIDS establishment or Nobel committee to acknowledge the significance of The Sammelweis Society giving their award this last year to National Academy member Peter Duesberg for initially alerting the scientific community as to the impossibility and destructive capacity of the “HIV=AIDS” hypothesis, and to appreciate the significance of the co-presentation of that award to investigative journalist, Celia Farber, for her initial expose regarding the iatrogenocide committed against gay men during the high-dose AZT era;

Z. And finally, the failure of the Promoters of AIDS, to address in any of our numerous invited all-expense-paid offers to hold a public forum, or media exchange, and respond to questions as to why not one of their more than 33 “HIV” test kits first initially patented and launched by Robert Gallo and Abbott Laboratories claim they can’t detect “HIV,” and why they continue to state on their package inserts after more than 20 years, that the significance of “HIV’s” molecular signature is not known, or that “HIV” cannot be detected or diagnosed using these methodologies.

These are the true ABC’s of AIDS denialism. Rather than advocating Abstinence, Being Faithful, and wearing Condoms as the ABC’s of AIDS, all of this information (and a mountain of evidence not presented here) points to the glaring absurdity that A (“HIV)” leads to B (immune suppression), which leads to C (AIDS). The viral hypothesis is far too simplistic to be supported a moment longer without immediate and intensive investigation of 17 other hypotheses which, in some cases, have led to reversion of acquired immune collapse by improving immune function, without the use of anti-retroviral therapies, or attacking the “HIV” molecular markers or T-cell counts.

It isn’t all bad news.

It is true that there have been some successes, and these should be intensively reappraised and investigated as well. For instance, Donald Rumsfeld’s former biotech company, Giliad Biosciences, makes the AIDS cocktail drug atripola, which is now making obscene amounts of money in a plethora of AIDS pogroms (as well as Giliad’s Tamiflu, purchased by the American taxpayer, to fight the global and non-existent “bird flu pandemic”).

It might also seem like progress is being made by some because George Bush, and his Global AIDS coordinator, Mark Dybul’s PEPFAR pogrom was funded by a propagandized and hoodwinked Congress, and will now move forward to continue to line the pockets of the pharmaceutical companies as they continue to dump their rank, non-safety-tested, in some cases failed cancer chemo-therapeutic cellular (AZT) poisons on millions of Africans, and other nations like India, China, and others, after drugs like nevirapine were withdrawn from use in the U.S. a few years ago because of its rank liver-destructive toxicity, especially in women, and are now continuously being dumped on Africans and others among the World’s most vulnerable.

Another piece of hopeful news is that Kevin de Cock who is a World AIDS leader, announced recently that “heterosexual AIDS is over,” except of course, and according to the WHO and to him, among large segments of Africa, and the African American community perhaps, who remain problematic not because of some difference compared to whites in their heterosexual behavior, but simply because they are black. Such institutionalized racism, cultural phobia, and targeted selective testing biases have come to define the current “AIDS pandemic.”

It also might be perceived by some as good news that because of the WHO’s ceaseless terror campaigns of “HIV/AIDS” paranoia, on the tourist visas of such nations as India, it is now declared that:


“Declaration to be made by applicants seeking to stay in India for more than one year:”

I hereby undertake that I shall subject myself to a medical test, including for AIDS within one month of arrival in India. In case I am found positive for AIDS, I will leave India,”

Or that even the BBC frequently runs stories like the following as recently as December 2008 (http://news.bbc.co.uk/2/hi/africa/7768059.stm):



Getting high on HIV drugs in South Africa

Alka Marwaha, BBC News

“Anti-retroviral drugs used to treat HIV/Aids are being bought and smoked by teenagers in South Africa to get high.”

“Reports suggest that the drugs are being sold by patients and even healthcare staff for money.”

“Schoolchildren have been spotted smoking the drugs, which are ground into powder and sometimes mixed with painkillers or marijuana.”

“Aids patients themselves have been found smoking the drugs instead of

taking them as prescribed.”

Or that Reuters at the beckoning call of public health officials newsflashes around the world stories such as the following without any self-critical awareness of even common sense:



On Thursday, Sep 21, 2006, as reported in Reuters, it was announced for the first time that the probable cause of increased “HIV’s infection had been found to be…..smoking cigarettes, but 9 of 10 other studies failed to find a link between smoking and progression to AIDS:

LONDON (Reuters) – “Smoking, already linked to several illnesses, may also increase the risk of infection with HIV, the virus that causes AIDS.”


“In a review of studies that looked at the association between smoking and HIV, British doctors said five of the six studies they analyzed showed smokers had a higher chance of becoming infected.”

“Nine of 10 other studies in the review that tracked the progression from HIV to AIDS found no link with smoking.”

“The studies identified in this systematic review indicate that while smoking might be independently associated with acquiring HIV infection, it does not appear to be related to progression to AIDS,” said Dr Andrew Furber, of the South East Sheffield Primary Care Trust.

“Furber and his colleagues, who reported the findings in the journal Sexually Transmitted Infections, said tobacco smoke may increase susceptibility to HIV infection by modifying a variety of immune system responses.”

As presented above, it is a hopeful idea that with a simple pen stroke, Mr. Obama could save billions of dollars to bail out our failing petrochemical economy that refuses to support the development of renewable energy, while it is being developed in civilized continents like South America and Europe. Such a pen stroke also could at the same time save many of our white 13 year-olds (like Ryan White who died of a liver bleed, and whose misfortune was exploited with the help of the moralistic Jesse Helms because he was a hemophiliac to advance the Ryan White Act during the Reagan administration). With the stroke of a pen, President Obama could even save the occasional “low risk” always faithful to her husband” soccer mom, boy-scout-leader dad, or world-class boxer like Tommy Morrison who tested “HIV” positive years ago but who now tests negative and whose career was ruined), or Arthur Ashe, or Kimberly Bergalis, or the Glasers, and countless others, from the devastation that will inevitably occur because of the universal testing now proposed by the CDC, the American Society of Pediatrics, the AMA, and other physician organizations to test everybody over the age of 13, everyone who enters an emergency room, the African American population of New York City, and of course, every infant born in a hospital. A pen stroke could save thousands of “low risk persons,” who will at low frequency, be convicted of being “HIV-positive” because they had a recent flu or hepatitis B vaccine.

If 2% of flu vaccine recipients, as Dr. Klausner from California writes in the New England Journal of Medicine, will express various “HIV” antibodies or epitopes (variants of molecules) after a flu vaccination in a country of 300,000,000 people, this means that six million people (2% of 300,000,000 people potentially could test positive for “HIV” antigens [Christian, P. Erickson, Todd McNiff, Jeffrey D. Klausner. Influenza Vaccination and False Positive HIV Results New England Journal of Medicine, Number 13 , Volume 354:1422-1423, March 30, 2006].

Josephson et al. (1) reported the results of a WESTERN Blot study that analyzed the assumed “HIV capsid protein” which is called p24. About the results of indeterminate patients (patients that aren’t either considered positive or negative), they claimed:

“Despite the fact the majority of p24-and p24/25-indeterminate specimens exhibited specific antibody reactivity with HIV core antigen (the P24 protein that supposedly forms the shell of the “HIV” virus), there was no evidence linking this reactivity to HIV infection. On the contrary, based on available data and limited patient information, we predict that HIV infection was not the cause in most cases…”

“A possible explanation for the specific p24 antibody reactivity in our patient population is that it represents cross-reactivity with another human retrovirus.”

HIV” was not the cause in MOST cases? In other words, these authors have claimed that although all of the samples tested were initially reactive, they “feel” that actuality none of the patients were really carrying “HIV,” because 12 were from “low-risk groups” (perhaps they weren’t black, or openly admitting that they had just experienced a 20 year addiction to heroin, crack or alcohol, or poppers, or were in fact pregnant, or had warts, or had a recent flu, hepatitis B, or tetanus vaccine, or had one of several dozen autoimmune diseases like arthritis that also generates “HIV-like DNA sequences” that are spuriously detected with PCR).

The explanation these AIDS researchers provided for the fact that none of the subjects who tested “HIV-positive were in fact “HIV-positive, is really quite imaginative. Perhaps these “HIV” test subjects were all infected with some other retrovirus to account for their indeterminate reactions (HTLV-5 is even suggested). So if you test indeterminate for “HIV” as all these 21 samples did, then perhaps you harbor “a different retrovirus,” than “HIV.” Perhaps in time, this “other retrovirus” will lucratively be non-specifically linked to 99 previously known and described human diseases, instead of the mundane 48 previously known diseases that “HIV” is said to cause currently?

But pathologists, or scientists that have invented tests for diseases might have a somewhat different world-view of disease definitions than other scientists or doctors?

For instance, these authors forgot to mention that that alcoholic hepatitis, exposure to alpha interferon therapy, the presence of antibodies of healthy patients with high affinity for polystyrene used in different test kits, anti-carbohydrate antibodies, anti-collagen antibodies, arthritis, systemic lupus erythematosus, scleroderma, connective tissue disease, dermatomyostitis, tuberculosis, some strains of malaria, hemophilia, hepatitis, hemodialysis, high levels of circulating immune complexes, herpes simplex I and II, HLA antibodies (to Class I and II leukocyte antigens), hyperbilirubinemia, hypergammaglobulinemia, leprosy, lipemic serum, malaria, the presence of some malignant neoplasms, mycobacteriaum avium, non-specific detection of free ribonucleoproteins, organ transplantation, other retroviruses, the receipt of gamma globulin or immune globulin (as prophylaxis against infections), multiple transfusions, pregnancy especially in multiparous women, Q-fever with associated hepatitis, primary billiary cirrhosis, primary sclerosing cholangitis, renal failure, rheumatoid arthritis, Stevens-Johnson syndrome, recent tetanus, flu, or hepatitis B vaccination, T-cell leukocyte antibodies, chronic drug addiction, or visceral leishmaniasis are all known to be responsible for a false-positive cross-reactive HIV test results, as all of these (and about 40 other) factors or disease syndromes have been shown to generate false “HIV” positives (2).

AIDS diagnosis is different in different places.

To further emphasize the deadly absurdity and unnecessary expense of the current situation and policies regarding “HIV/AIDS,” if someone “HIV-positive” is diagnosed with AIDS in the U.S., all he/she has to do is go to Canada for another opinion, and voila (!), nineteen out of twenty times he/she will no longer be an AIDS patient, and thus not be prescribed the toxic drugs that actually bring mortality to slightly under half the recipients because of their “side effects,” such as liver or pancreatic failure, neurological damage, lymphoma, mitochondria destruction, and heart attacks, all typically requiring millions of dollars for further palliative treatments.

One must also take into account, the colossal waste of lives and money that continues to occur because of the misguided and illogical surveillance definitions of “HIV/AIDS are different in different places. The standards for making an “HIV,” “ARC,” or “AIDS” diagnosis vary, and are different depending upon the country or region of a country where people are tested.

In 2001, the CDC’s MMWR, Guidelines for Laboratory Test Result Reporting of Human Immunodeficiency Virus Type 1 Ribonucleic Acid Determination, the recommendations from a CDC Working Group as of November 16, 2001 (50(RR20);1-12) reported that:

“Results obtained with available test methods are variable, and laboratories present these results in different ways, indicating that guidelines to promote standard practice in reporting of test results are warranted.”

“No test reporting standardization exists; specifically, standard units of measurement of test methods have not been established. Laboratory viral load test reports should be accurate and adequate for patient treatment and public health monitoring of the HIV and acquired immunodeficiency syndrome (AIDS) epidemic. To assure test reporting comparability among laboratories, standard methods are needed; moreover, standardized results are needed for early detection of infection, early access to patient care, and early detection of treatment failure.

In January 1, 2000, the CDC HIV-infection surveillance case definition was expanded to include viral load test results despite the fact that:

 “In certain cases, laboratory slips indicated that HIV had been detected at a value below the test’s lower limit (e.g., HIV detected was <400 copies/mL), or the laboratory slip provided an actual number of copies outside of the stated reportable range.”

 The National Institutes of Health and Henry J. Kaiser Foundation, US Department of Health and Human Services, National Institutes of Health, 2001 have claimed the following (available at <http://www.hivatis.org&gt;. Accessed July 20, 2001):

 .”Until a common standard is available to use for normalizing values obtained with different assay methods, choosing one assay method is advisible when HIV RNA levels are monitored to guide therapeutic decision-making. The goal to develop a common standard for normalizing values obtained with different test kits has recently been reported.”

 “Available tests are not licensed for diagnosing HIV infection, but the viral load test results are used for reporting HIV infection to local and state health departments.”

 This was the state of affairs at the beginning of our new century. Things have become much worse since then.

 In May 2000, President Clinton declared AIDS to be a national security threat. President Bush and Congress have spent huge funds for America, Africa, and elsewhere. In October 2008 $48 billion was given for Africa alone, to be spread over the next five years. Being directly under the purview of his executive office, and commandeering almost 1% of the entire Federal budget, it is imperative President Obama order a complete review of both the incredulously higher U.S. death rate compared to all other Western nations, and ascertain the truth of genuine African AID belatedly acknowledged by many authorities to be grossly overestimated.

United States annual “AIDS deaths” have been near 16,000 for many years, providing fodder for ceaseless news accounts. What is keep quiet however, and is censored may be more accurate, is that the U.S. death rate is twenty-five times European Union (EU) country citizens’ death rate. Other countries such as Canada right next door, Australia, and New Zealand, match the EU success, all with AIDS deaths having sunk to double digits, basically to background levels before the term AIDS was coined 24 years ago. The self-perpetuating U.S. death toll springs from an errant definition of AIDS employed solely in the U.S. that initiates toxic drug therapies, that in turn brings iatrogenic AIDS mortality, with U.S. health generals inexplicitly failing to learn from these resounding “successes.” Having demonstrated their commitment to their undeviating tragic course, it is up to the President to relieve the captain and officers of the leviathan U.S. AIDS War and “Challenger-sized disaster,” without a moment’s delay.

Moreover, the preventable U.S. death toll, compared to success elsewhere, should be succinctly and logically explained to the public, politicians, and medical professionals. Equally important, there are many scientists with world-class credentials able to enlighten President Obama, and President Obama’s administration must allow them to be heard and not filtered through layers of handlers, or the chiefs of NIH unwilling to review the truth amply shown all around the world.

Since other Western countries attribute a low WBC count as only an AIDS indicator “illness,” and despite having similar “HIV-positive” rates, other Western countries have but a tiny fraction of the U.S. AIDS diagnoses. Thus, few are given the toxic treatment drugs designed to raise the WBC count and suppress “viral load.” Perhaps the physicians of these countries realize that low WBC and CD4+ T lymphocyte counts (CD4 counts) are associated with a variety of conditions, including many viral infections, bacterial infections, parasitic infections, sepsis, tuberculosis, coccidioidomycosis, burns, trauma, intravenous injections of foreign proteins, malnutrition, over-exercising, pregnancy, corticosteroid use, normal daily variation, psychological stress, and social isolation? There are also a number of people (about 3-5% of non-“HIV-positives) who are completely healthy and who have low CD4 counts for no apparent reason.

However, in the U.S., even if and when WBC counts are optimistically raised, patients’ clinical results and mortality have been the opposite. The results have been consistent annual U.S. AIDS cases of 39,000, with the high death toll already noted, clearly tied to the taking of treatment drugs. These deaths are usually blamed as “HIV-caused” even though scientists after 25 years on their medical Manhattan Project have never been able to elucidate how “HIV” actually kills cells, and have never been able to provide an electron microscope photo from the tissues or blood of an “HIV-positive” or “AIDS patient,” of such cellular murder. As for the real facts, minimal deaths in all other countries resoundingly implicate the U.S. AIDS definition and consequent treatment drugs:

Germany with a population of 83 million had 73 “AIDS deaths” in 2006 and but 373 new “AIDS cases;” other 2006 examples include Sweden with 9 million having but 10 “deaths” and 59 new “AIDS cases,” Canada with 33 million people had but 34 “deaths” and 255 new “AIDS cases,” and so on (Sources: EUROHIV; HIV/AIDS surveillance in Europe: end-year report 2006; Surveillance Report to Dec. 31, 2006, Public Health Agency of Canada).

In 2006, the December Senate approved Burr’s bioterrorism bill-a bill just in time for Christmas to establish the Biomedical Advanced Research and Development Authority, commonly referred to as BARDA, which passed by unanimous consent. The bill describes how forced vaccines, quarantines, criminalization of “HIV” and other molecular diseases should be signed into law as the ‘debate’ regarding Bush’s war in Iraq and Afghanistan continued. Also in 2006, the periodic and massive recalls of “HIV-test kits” continued. For example, the FDA recalled Vironostika HIV-1 test kit lots: 259606, 121566,1008926, 259606, 121567, 1008926, 259606,121568, 1008926, 259605, 259717,160342, 1011220, 259605, 259717,160339, 1011021, and said:

 “These HIV-1 finished kit lots in the field have been reported to contain EnzAbody reagent that appears noticeably cloudy and/or flocculent, instead of clear and non-turbid as expected 30 minutes after reconstitution. Use of cloudy EnzAbody could possibly increase your risk of inaccurate HIV test results in patients and therefore should be avoided.”

A more of less complete list of similar recalls would take volumes of tables to present here, including reports regarding “HIV-rapid test” banning and confuscation by the FDA because of fraudulent claims, and are available upon request of the reader.

Suffice it to say, those accused by these kits of being “HIV” positive are rarely informed about “HIV” testing accuracy as they prepare themselves for eventual death or perpetual toxic drugging, amounting to approximately $350,000/patient over the course of their “treatment” according to recent reports. For instance, in 2006, Dr. Bruce R. Schackman, chief of health policy at Cornell’s Medical College in New York and lead author of a paper appearing in Medical Care in 2006, a journal published by the American Public Health Association claimed that “…patients can live average 24 years, if they pay $385,000.

 But there are legions of “HIV-positive” individuals who have escaped the State’s reporting systems as well, who have never taken or who refused to take “HIV” tests or anti-retrovirals, and who also have lived for 24 years since the beginning of the AIDS era. The self-reported reasons for their disease-free and drug-free survival has been amply documented on documentaries that have won awards such as Special Jury Prize at the AFI Los Angeles International Film Festival (e.g. “The Other Side of AIDS”).

In 2006, a nationwide team of AIDS researchers led by doctors Benigno Rodriguez and Michael Lederman of Case Western Reserve University in Cleveland disputed the value of viral load tests-a standard used since 1996 to predict when progression to AIDS would occur in drug-naïve individuals, and to grant approval to new AIDS drugs, after their study of 2,800 HIV positives concluded that viral load measures failed in more than 90% of cases to predict or explain immune status (3):

 Viral load is only able to predict progression to disease in 4% to 6% of HIV-positives studied, challenging much of the basis for current AIDS science and treatment policy.”

And a major groundbreaking study affirms being “HIV-positive,” by itself, is anything but a death sentence: Denmark’s Dr. Nicolia Lohse, with seven co-investigators, published Jan. 16, 2007 in the Annals of Internal Medicine the status of every “HIV-positive” Denmark resident through the years 1995-2005. The study compared those “HIV-positive” to otherwise normal Danes, and tallied mortality of any cause, including drug overdose, accident, alcoholism, AIDS, etc. The study concluded that the life expectancy of Danes judged “HIV-positive” at age 25, taking the milder anti-viral drugs started in 2000, could expect another 39 years of life to age 64, compared to otherwise normal Dane life expectancy of 76.

What is even more striking in Lohse’s charts is that 25% of those “HIV-positive” in the years’ 2000-2005 cohort refused anti-viral drugs, yet had even lower mortality rates compared to the anti-“HIV” drug takers, the “HIV-positive” women refusing drug treatments in particular approaching a normal life span. Importantly, Lohse also states those “HIV-positives” typically had high health risks such as smoking, alcohol and other drug addiction, so it should hardly be surprising those “HIV-positive” would have a shorter life span than fellow Danes (who have less alcohol-related accidents, drug overdoses, or lung cancer/heart mortality).

Lohse’s Denmark study, as do countless others, demolishes the entrenched belief that having “HIV” antibodies demands anti-viral treatments. In fact the opposite is true, as his study clearly documented the extraordinarily high mortality of those taking the earlier anti-virals like AZT. Again, these results emphatically contradict the belief that “HIV-positives” progress to deadly disease, unless anti-viral/retroviral drugs are taken for significant lengths of time.

In view of the Denmark study being published in January, 2007, in a major journal of wide readership and undoubtedly delivered to thousands of scientists’ offices including the CDC and the National Institute of Allergies and Infectious Disease (NIAID), and also considering the low AIDS mortality enumerated for years in Europe’s annual surveillance reports (available at the click of a mouse), the ignorance and continuance of big pharma-directed polices of the Global and U.S. health generals are appalling and unforgivable. As to the American press, their self-censoring of Europe’s success (and Canada, Australia, and so on), and nonstop promoting of HIV hysteria, speaks for itself.

President Obama and Congressional leaders must demand of NIAID Director Dr. Anthony Fauci, having vainly been sent billions of dollars to understand “HIV,” why he has not read Lohse’s study, or not learned from the other countries’ such as Germany’s remarkable success over AIDS mortality.

The utter failure of NIAID cannot be tolerated for a day longer. For 20 years under Dr. Fauci, NIAID has directed hundreds of chemotherapeutic anti-HIV trials strictly limited to a first set of anti-viral/retroviral chemicals tested against a second set of toxic chemicals, without ever permitting a single true non-toxic placebo given to human subjects since the 1987 Fischl AZT trail, that was terminated prematurely, became un-blinded, had many sentences of medical records blacked out before the data was submitted due to a Freedom of Information Act Request. Patients in the AZT-treated arm were given transfusions to stay alive until all of them were placed on “the life saving medication” and eventually all died 3 years into the trail. (See John Lauritsen’s books, ‘The AIDS War; Propaganda, profiteering and genocide from the medical-industrial complex’ (1993); and ‘Poison by Prescription; The AZT Story’ (1990); ‘The AIDS Cult’ (1997), and his essays entitled, ‘AZT on Trial’ (1987),’AZT and Cancer’ (1989),’The AIDS War’ (1991), ‘HIV Voodoo From Burroughs-Wellcome’ (1991),’FDA Has Second Thoughts on AZT’ (1991),’FDA Documents Show Fraud in AZT Trials’ (1992),’Looking Back on Berlin’ (1993),’Recovery from “AIDS”‘ (1993),’The Death of Rudolf Nureyev’ (1993), and ‘The Poppers-Kaposi’s Sarcoma Connection’ (1994). Also see investigative journalist, Celia Ingrid Farber’s damning essay, entitled, ‘Sins of Omission; The AZT scandal’ (1989).

The “Concorde” trial, a collaborative effort between researchers in the United Kingdom and in France, and the U.S. Veterans Administration’s Study 298, both compared early and delayed AZT treatment, but the Concorde study researched asymptomatic patients at all CD4 levels, while the VA study included only symptomatic patients with CD4 levels of 200 to 500.

In 1992, The Veterans Affairs Co-operative Study Group reported that AZT disproportionately harmed Blacks and Hispanics, and provided no benefit to the quelling of advancing immune suppression in Caucasians, and harmed healthier subjects (early treated) more than persons considered to exhibit clinical symptoms of AIDS [JD Hamilton et. al. and the Veterans Affairs Cooperative Study Group. ‘A controlled trial of early versus late treatment with zidovudine in symptomatic human immunodifficiency virus infection.’ New England Journal of Medicine, 326: 437-434, 1992], and in 1994, the Concorde study, which was up until then the longest, largest, and most carefully controlled AZT trail reported:

“The results of Concorde do not encourage the early use of zidovudine in symptom-free HIV-infected adults. They also call into question the uncritical use of CD4 cell counts as a surrogate endpoint for assessment of benefit from long-term antiretroviral therapy” [Seligmann et al., Concorde: MRC/ANRS randomised double-blind controlled trial of immediate and deferred zidovudine in symptom-free HIV infection. Concorde Coordinating Committee. Lancet, Apr 9;343(8902):871-81, 1994].

The same kind of results obtained during these massive human drugging experiments was further emphasized after the first decade of HAART therapy where it was reported that:

Methods: We analyzed data from 22,217 treatment-naïve HIV-1-infected adults who had started HAART and were followed in one of 12 cohort studies. The probability of reaching 500 or less HIV-1 RNA copies per mL by 6 months, and the change in CD4 cell counts, were analyzed for patients starting HAART in 1995-96, 1997, 1998, 1999, 2000, 2001, and 2002-03. The primary endpoints were the hazard ratios for AIDS and for death from all causes in the first year of HAART, which were estimated using Cox regression.

Interpretation: Virological response after starting HAART improved over calendar years, but such improvement has not translated into a decrease in mortality. [The Antiretroviral Therapy (ART) cohort Collaboration-www.thelancet.com Vol 368, 451-58, August 5, 2006].

This comes as no surprise. Dr. Fauci’s AIDS promotionalism began even before the AIDS era when he claimed that immune suppression is caused by doctors!

(The term AIDS promotionalism or Promoters of AIDS is being used here to indicate pharmaceutically myopic doctors like Fauci, who continue to advance the “HIV”=AIDS=Drug=Death paradigm against all the evidence which is obtainable in the mainstream scientific literature such as The Lancet, and the New England Journal of Medicine). Doctors cause immune suppression, Fauci claimed, if they subject their patients to multiple transfusions, transplant surgery, or corticosteroid administration, as these drugs and treatments can non-specifically induce AIDS-specific drops in T-cells with high frequency (4, 5). Fibrosis of the lung due to heavy crack cocaine use also was considered a potent inducer of the AIDS-defining illness PCP, by Fauci and many other “doctors” before the AIDS era. The expense to the American taxpayer of this iatrogenic carnage cannot even be calculated, but it would amount to a staggering amount of money that could by itself restore the American economy.

Perhaps reminder of a similar health catastrophe in recent history might here be presented in order to illustrate the potential magnitude of continuing to support the widespread drugging of persons throughout the world without sufficient time and study to determine how these rank poisons and toxins could affect millions of people.

What a difference it would make for our society and world under the Obama administration to direct public health policy not predicated upon failed and dangerous Pharma-doctor-prescribed and junk-science-based pogroms like those of the past that were predicated on fears, racism, homophobia, sexism, faith-based recommendations, or worse, as it was the case with The Church of Medicine’s love affair with DES:

National Academy. Dr. Bern had exposed the dangers of DES (diethylstilboestrol)- a widely used synthetic oestrogen (Modified from DES action of Australia http://www.desaction.org.au/aboutdes.htm):

Synthesised in 1938, laboratory studies showed animals administered DES developed mammary cancer, with high rates of fetal death, sterility and cancer in the offspring. Despite this, DES was approved for use in humans in 1940. Initially used to treat late pregnancy complications, by the mid 1940s the use was widened to include the prevention of miscarriage, i.e. for prophylactic use by women who had a history of miscarriage. However by 1949 DES was seen as making “a normal gestation more normal”. By the early 1950s DES was being prescribed and marketed as a general pregnancy “tonic”, mixed with vitamins and recommended for all pregnant women to ensure healthier pregnancies with “bigger and stronger” babies. A 1953 study showed, to the surprise of the researchers, that the DES-treated group experienced higher rates of miscarriage, premature labour and neonatal death than the control group, and the 1953 Dieckmann study was ignored. DES was already entrenched as standard obstetric clinical practice. Also by this stage DES was being aggressively promoted by the drug companies for use in all pregnancies. In 1971 it was discovered that DES caused clear cell cancer of the vagina/cervix in DES daughters. DES was thus proven to be carcinogenic in humans. Regardless of these findings, it was continued to be used as a treatment for acne, to dry up breast milk, as a contraceptive like a morning after pill, as hormone replacement therapy during menopause, as a treatment for “tall girls” to stunt their adult height, and to fatten up livestock to increase profits.”

In 1981 landmark publication, ‘Developmental Effects of DES in Pregnancy’ was edited by Arthur L Herbst and Howard A. Bern, which brought together leading experimental researchers and expert clinicians on DES. In an experiment on mice, Herbst and Bern showed that in later life, the immune system of DES exposed mice was suddenly compromised. Preliminary studies of DES daughters in the early 1980s indicated that DES exposure is linked with immune system problems, including a higher incidence of autoimmune disease, such as asthma, arthritis, diabetes, systemic lupus and thyroid dysfunction.”

Lohse’s recent study, The Concorde and Veterans Affairs studies, and many others in the peer-reviewed literature that have published similar results, have drawn back this forbidden-placebo curtain with spectacular life expectancy revelations. Denmark’s study, buttressed by countries’ successes already enumerated, demands an American about face, to employ the scientific method not permitted for two decades. A thorough review by President Obama is also relevant in the most fundamental fiscal way, as spending on AIDS now comprises almost 1 percent of the entire federal budget, and at this point constitutes a complete waste of taxpayer monies, and worse.

As for Africa, news stories have leaked out from WHO officials admitting AIDS numbers had been greatly exaggerated beginning in the 1980’s, and even most medical “experts” in the U.S. never have realized that most all African AIDS cases came from the “Bangui Definition,” created in 1985 by WHO and CDC officials meeting in the Central African Republic capital city of Bangui 23 years ago.

This Bangui definition stated an African AIDS case was anybody having the three health conditions of a fever, weight loss, and diarrhea over the period of 30 days (or a cough instead of fever), requiring no medical tests whatsoever that would, if taken, typically be diagnosed as malaria, tuberculosis or scores of other identifiable, treatable diseases.

Instead, virtually all African disease mortality was wrongly piled together as “AIDS” and still is.

Until recently, in Africa, and due to a lack of medical infrastructure in many areas, persistent war in other regions, and the ravages of that continue because of Apartheid in South Africa, positive “HIV-AIDS” diagnoses have been made traditionally without the use of serological testing altogether, if the treating physician felt that a particular case of persistent diarrhea or persistent coughing or tuberculosis exhibited by their patients appeared to be an AIDS case. In addition, many of the cases of “AIDS” in Africa that the WHO uses for their dire estimates of 40 million infected were extrapolated from maternity clinics or with non-validated rapid tests, and in many cases collected only from where women are tested, and projected onto the rest of the population, who weren’t pregnant (pregnancy itself is a known reason for false positives, according to the CDC). Therefore, the AIDS-defining illnesses may be different among Africans than among non-Africans, may differ among African males and females, and are different among those tested by the Red Cross, from those tested by the CDC. These differences in testing standards make it possible to test positive in the United States in the morning, and by flying to Canada and getting tested, one can test negative in the afternoon.

For twenty years, news stories, politicians, and drug companies exploited fear, promoting massive African AIDS cases that, in fact, never did exist, with “AIDS” cases bearing no relationship to immune deficiency caused by a virus. It is vital to further note that most of the $9.8 billion a year of U.S. tax dollars presently going to Africa (noted above) via PEPFAR (President’s Emergency Plan For AIDS Relief) is earmarked for the exact anti-viral drugs that have been causing the mortality of Americans the last 15 years, year after year, and with no solution in sight staying the present course.

But these facts have not deterred PEPFAR-in fact they have provided it’s justification. Instead of critical examination of evidence, policies based on biblical rather than scientific evidence have been advanced. Noted and celebrated researchers such as Robert Bailey of the University of Illinois are given lavish praise for now promoting such ideas that all African males should be circumcised to prevent the spread of the dreaded “virus,” “HIV.” It is beyond belief and human reason that these pogroms based on these bizarre interpretations of reality continue to claim that Egyptian and Hebrew biblical practices such as circumcision has won out over pharmaceutical technologies, vaccines, microbicide campaigns, and breast feeding dissuasion campaigns designed to diminish Mother-To-Child Transmission of “HIV” (each of which except circumcision have been halted by various oversight and human protection committees recently due to their tendency to increase rather than decrease “HIV” incidence in these various African test populations). But a new AIDS ambassador might well ask if it is true that biblical approaches such as circumcision really reduces “HIV” incidence of African men in STD clinics typically presenting with multiple STD’s and genital ulcers simply because AZT, HAART, microbicides, vaccines, breast feeding dissuasion campaigns, nevirapine, and condom crusades simply haven’t worked, and bizarrely, have each, except for circumcision, have increased the level of morbidity in African “lab rats” before they were halted.

If you read Pasteur and believe in the “germ theory,” the failure to seroconvert to a positive “HIV” test result after 63 failed “HIV” vaccination trials which include even booster series (two or more vaccinations given sequentially) means that, the principles underlying immunology, biochemistry, genetics, epidemiology, virology, cell biology, pharmacology, neonatology, and cancer biology don’t apply to “HIV/AIDS,” or it means that the hypothesized “HIV” causation of “AIDS,” and the imagined molecular biological causal basis of AIDS and several other “molecular diseases,” whose science the AIDS era was based upon, have generated catastrophic disasters that require our immediate attention so that we can revise these vehemently defended deadly, and expensive Public Health policies that continue to ruin the lives of millions.

It is not accidental, that all of these “molecular diseases” that are in question are those syndromes, and their molecular markers, that have had some connection with cancer, and which have been blamed on such processes as supernatural abilities to mutate, “oncogenes,” “retroviruses,” the enzyme reverse transcriptase, and the concept of “slow viruses.”

Proof of the logic of this statement in the context of “HIV/AIDS” is simply realized by considering the contradiction regarding how some 33 million people in the world that are said to be “infected” and walking around with specific molecules of “HIV” in their bodies, or with molecules generated by the body’s response to “HIV,” while only 24/741 or 19/672 “HIV” vaccinated individuals seroconverted after vaccination in the recent STEP trials. Those few who appeared to have produced antibodies to “HIV” after vaccination, or even after two vaccinations as the recently aborted “HIV” STEP trials demonstrated, and in the 62 prior “HIV” vaccine trials, couldn’t even show appropriate T-cell responses in most cases as evidence that something foreign had been injected into their bodies. The control group not given “HIV” molecules had less “seroconversion” in response to their control injection, while the “HIV-component” vaccinated showed slightly more “HIV-positive” signals, but the differences between the two groups weren’t significant. But to erect a smoke screen to cover this massive blunder, it was a result interpreted and fed to the press by the promoters of AIDS as evidence that the “HIV-vaccinated” went out for some reason and had more “risky” behaviors than the control vaccinated group, which is why the trial was aborted, or that they had more susceptibility to the worthless adenoviral vector used as a carrier, or that they had “more or fatter” cellular targets for “HIV” to “infect.”

It should be emphasized that this Step Trail result is why one of the AIDS Czars, Anthony Fauci, cancelled the so-called upcoming PAVE “HIV” vaccine trial, until the scientific basis for the “HIV/AIDS” paradigm is re-examined. The failure of another large millions of dollar AIDSVAX vaccine trail a few years ago, also forced Robert Gallo to publish in the form of damage control letters in the world famous Science magazine, that a sound rationale for future vaccine trials is needed before trials should continue, and before the public’s faith in vaccines and AIDS paradigm (and gargantuan AIDS funding) is undermined by repeated vaccine trial failures. Dr. Gallo even compared the failure of the STEP trial to ‘The Challenger disaster.’

Considering the failure of no less than the 64 previous “HIV” vaccine trials that are on the record (and which can be provided upon request to the reader), and the “Challenger-sized” disaster claimed for the recently aborted STEP trial that was discontinued because it increased, rather than decreased the rate of acquiring an “HIV-positive” test results among the vaccinated, isn’t it about time that we heed Dr. Fauci’s and Dr. Gallo’s suggestions to re-examine the entire basis of “HIV/AIDS” “science?” Or should we continue to reward failure after failure, as was horrifically demonstrated a few years ago when the promoters of AIDS asked for, and received a $870 million dollar taxpayer-provided gift (Donald Francis’s VAXGEN) to dump their failed “HIV” AIDSVAX vaccine program, and then manufacture an even more problematic, ineffective, and dangerous anthrax vaccine in the name of National Security? Although many of the 63 recorded “HIV” vaccine trials were smaller than these colossal disasters and wasting of our tax dollars just mentioned, they do add up, and then often usher in even more no-bid contracts totaling in the billions for continued failure.

The available evidence suggests that the simplest explanation for the vaccination failures and other failures of the “HIV=AIDS=Death paradigm is that the injected components of “HIV,” and the antibody responses to these components, have nothing to do with a virus that is foreign to the human body, or which is infectious, or which should be aggressively treated with toxic drugs. This evidence also suggests that there are clearly identifiable reasons to explain why one of the leading causes of AIDS death today in the United States of America are iatrogenic (doctor-induced) deaths, and that the leading cause of death among “AIDS patients” has come to be liver failure, heart attacks, gastroenteritis, anemias, kidney failure, infectious diseases that form bacterial biofilms, and other non-AIDS-defining syndromes.

 PART II: The slippery slope of institutionalized racism and homophobia, and what are the probable causes of “HIV?”

It is easy to find fault with Public Health policies, but can the iatrogenic and pharmaceutical carnage that is currently occurring in the name of “HIV/AIDS” be replaced by a more rational science, and effective public policy? As with any science based on an assumption, it is that assumption, and typically the origin of that assumption, that is baseless. For example, a likely explanation of the origin of “HIV” should derive not from notions of monkey-to human or ape-to human transmission of a virus due to Africans smearing monkey blood on their loins for sexual orgies as published in The Lancet and other top journals at the beginning of the AIDS era (6), nor was “HIV” likely transmitted to African children or their parents by playing with or by eating dead monkeys or chimps as “bush-meat” because their parents couldn’t find or afford toys or food (7), or by Africans in Cameroon building cities 125 years ago and having “close associations with chimps” as was published this last October 2008 in Nature and news-flashed around the world. As recently as this last year, for instance, and in no less a journal than Nature we find that, unfortunately, Western racism continues:

2008, October. News Flash: “HIV/AIDS Originated 125 Years Ago, Spread

from Chimps to Humans…”

“TUCSON, Arizona, October 2, 2008 (ENS) –

New research indicates that the most pervasive global strain of HIV began spreading among humans as early as 1884, suggesting that growing urbanization in colonial Africa through the early 1900s set the stage for the current HIV/AIDS pandemic. More than 25 million people have died of AIDS since 1981, and at least 30 million people are living with the disease today.”

The estimated period of origin, much earlier than the previous estimate of 1930, coincides with the establishment and rise of urban centers in West-Central Africa where the pandemic HIV strain, HIV-1 group M, emerged.”

The growth of cities and associated high-risk behaviors may have been the key change that allowed the virus to flourish, scientists believe.”

The research, led by Michael Worobey, an assistant professor of ecology and evolutionary biology at the University of Arizona in Tucson, was co-sponsored by The National Institute of Allergy and Infectious Diseases, part of the National Institutes of Health, and the David and Lucile Packard Foundation. The findings are published in the current issue of the journal Nature.”

Research shows that the HIV virus spread from chimps to humans in Southeastern Cameroon. Worobey said the resulting HIV epidemic among humans correlates to the growth of urban centers near this area, principally the present-day city of Kinshasa in the Democratic Republic of the Congo, which began as a colonial center for Belgium.”

What research? Flawed and highly suspect comparisons of chimp and human “HIV” sequences? Although out of political correctness more than anything else, this 2008 racist Nature-published interpretation of “HIV” spreading from chimps to humans in the vicinity of Cameroon isn’t as clearly spelled out as it was when Robert Gallo and his colleagues promoted the idea that their first human and mistaken “cancer retrovirus,” “HTLV-1,” was spread during the “slave trade,” and which according to them also was associated with black people and monkeys during the transcontinental forced slavery and transport of 100 million Africans and monkeys Centuries ago. It is far more likely, because of modern molecular evidence, that “HIV” originated not from Africans, African Americans, Haitians, African-non-human primate “associations,” or from anyone else harboring a lot of melanocytes or who lives close geographically to non-human primates (“HTLV-1 was the so-called first human cancer retrovirus upon whose isolation and epidemiological techniques and assumptions the “HTLV-III/”HIV” paradigm was based).

It was in this context, and disturbingly, HTLV-III (later called “HIV-1″) was introduced at a press conference given by Margaret Heckler and Robert Gallo, as being “a variant of a known human cancer virus,” and it was announced that “HTLV-III” was “the probable cause of AIDS.” In May, and in the months following the press release, four papers from Dr. Gallo’s laboratory claiming that the HTLV-III retrovirus was the cause of AIDS were published in Science. These are the 4 papers that established that “HIV’s” signature was present in 1/3 of “AIDS patients” rather than associated with serum samples from a few ARC and a Kaposi’s patient (as Montagnier and Barre-Sinoussi’s work the year before had claimed), and these papers supposedly constituted the experimental basis of the hypothesis that “HIV” causes “AIDS.”

The initial description and press release by Margaret Heckler of “HIV” as a known variant of a cancer virus in retrospect was perhaps unfortunate, because as stated by Gallo and Montagnier in a 2003 New England Journal of Medicine article (Gallo RC., Montagnier LM. The discovery of HIV as the cause of AIDS. NEJM Vol. 349:2283-2285, December 2003), the molecular signature that was associated with “HIV,” was derived from the same techniques used to characterize the molecular signatures of Gallo’s previous putative cancer viruses, “HTLV-I” and “HTLV-II.” But the team claimed that “HTLV-III (“HIV”) was distinctly different from Gallo’s leukemia-associated retroviral signatures of “HTLV-I” and “HTLV-II” in that it attacked and killed lymphocytes, rather than induced them to proliferate as would leukemic cells.

The patent and intellectual property issues of the U.S. and French governments surrounding the creation of an “AIDS” blood test to protect the blood supply also proved to be an unfortunate series of confusing and hateful events that made possible accusations against the American-French collaboration that virtually insured suspicion of the “HIV=AIDS” hypothesis, and served, it could be argued, to derail progress toward defining AIDS and immune suppression.

Gallo becomes a shepherd and scientific peer review is avoided:

Although it was claimed that there was an initial molecular concordance between the Montagnier’s “LAV” and Gallo’s “HIV” molecules discovered “in isolates” from what were then defined as ARC or AIDS patients, and despite a laboratory accident, whereby it was claimed that a different contaminated isolate was sent from The Pasteur Institute to Bethesda which “infected” the cultures of the Gallo group, Robin Weiss’s group in England, and at least several other laboratories, research in these labs was thus confounded for at least the next year (Robert Gallo, personal communication). And despite the fact that Gallo shepherded through the 1983 Barre-Sinnousi et al. paper through to publication because it didn’t pass initial review, the American-French collaboration, the patent on the blood test was filed by Dr. Gallo the day of the press release. This patent filing was from henceforward destined to be plagued by numerous accusations of stealing, deception, misconduct, and fraud. However, shepherding of papers occurs frequently among cooperating scientific groups, and as such, such papers are not rigorously “peer-reviewed.” The interests of the U.S. and French governments regarding the patenting of an a blood test, fear of contagion, religious views regarding morality, sex, and drugs, and politics began an era that perhaps should be characterized as the most politicized disease in history. And even though it was finally concluded that there was no misconduct or steeling found in the Gallo/Papovic case, the numerous accusations, and the lengthy and costly investigations of misconduct and blame only served to polarize those involved and those critical of them, and the charges of wrong doing only fueled bitter rivalry and criticism that has lasted for more than two decades.

The Gonda letter said there was no “HIV,” HTLV-I,” “HTLV-II,” or “HTLV-III in the Papovic/Gallo samples:

The molecular markers that became known as “HIV” in isolates that Montagnier’s and Gallo’s group tied to a clinical syndrome in “pre-AIDS” and AIDS patients and in one putative ARC patient as the Pasteur had intensively studied (Patient One), constituted a strong motive for many to pursue in depth, the nature and meaning of that molecular signature. Then, in November, of 1984, the Pasteur Institute investigators published “the genetic sequence” of “LAV.” Many, however, were critical of the strength of Montagnier’s and Gallo’s correlation between “HIV’s” molecular markers and the disease syndromes to which these signatures were associated because of documents that became available during the numerous investigations such as the following, by the Head of the Electron Microscopy Lab at the NCI. The document is significant because it was apparently sent one month before the Heckler-Gallo Press release and the Papovic/Gallo et al., publications in Science, as provided by the investigative medical journalist, Janine Roberts:

Dr. Mica Papovic March 26, 1984 (Received March 27):

Laboratory of Tumor Biology


Building 37, Room 6B22

Bethesda, Maryland, 20205

Dear Mica

I am sending you 4 extra copies of results requested by Betsy Read. She

said Dr. Gallo wanted these micrographs for publication because they con-

tained HTLV particles. If this assumption is based on the cultures being

antigen positive, I would like to point out that the “particles” in micro-

graph 0905 are in debris of a degenerated cell. No other extracellular

virus-like particles” were observed free between cells anywhere in the

pellet. The small extracellular vesicles in 0904 are at least 50% smaller

than HTLV mature particles seen in type I, II, or III. Again, these vesi-

cles can be found in any cell pellet. I do not believe any of the particles

photographed are HTLV I, II, or III.

cc Robert Gallo Best regards,

Betsy Read Mathew Gonda, Ph.D

Head, Electron Microscopy Laboratory

Gallo’s “36% Causality:” “HIV” causes AIDS!

An articulate analysis of these and other criticisms have been recently presented in retrospect by The New AIDS Review (http://www.newaidsreview.org/blog/index.phphas), especially the data presented in Gallo’s May 4th Science paper (Science May 4 2004, Frequent detection and isolation of cytopathic retrovirsuses (HTLV-III) from patients with AIDS and at risk for AIDS…”) about which the Gonda letter above shown failed to identify any viruses:

Gallo didn’t find HIV in 48 out of samples from 48 AIDS patients, as he keeps implying. He found whatever he took as the signature or signal for it in 26 out of 72 samples of AIDS patients, which is rather different 36%. The rest of the 48 came from pre-AIDS patients (who may have had nothing but a cold) and three mothers (who were perfectly well) and 1 clinically well gay man out of 22 (who later developed AIDS). 119 in total from which 48 scored positive.”

As reviewed by Michael Geiger, Executive Director of HEAL San Diego (Health Education AIDS Liason):

Robert Gallo’s claim that HIV is the cause of AIDS was first put forward on the basis of four papers he published in Science; May 4 1984 issue. (Popovic M, Sarngadharan MG, Read E, et al. Detection, Isolation, and Continuous Production of Cytopathic Retroviruses (HTLV-III) from Patients with AIDS and Pre-AIDS. Science 1984;224:497-500.; Gallo RC, Salahuddin SZ, Popovic M, et al. Frequent Detection and Isolation of Cytopathic Retroviruses (HTLV-III) from Patients with AIDS and at Risk for AIDS. Science 1984;224:500-502; Schupbach J, Popovic M, Gilden RV, et al. Serological analysis of a Subgroup of Human T-Lymphotrophic Retroviruses (HTLV-III) Associated with AIDS. Science 1984;224:503-505; Sarngadharan MG, Popovic M,Bruch L, et al. Antibodies Reactive to Human T-Lymphotrophic Retroviruses (HTLV-III) in the Serum of Patients with AIDS. Science 1984:224:506-508).”

A press conference at the time, arranged and conducted by Margaret Heckler, then Secretary of Health and Human Services for the Reagan administration, claimed that his work in the soon-to-be-published papers had shown that HIV was the “probable” cause of AIDS.”

The New York Times (EIS-Epidemiology Intelligence Service trainee) Lawrence K. Altman) reported the news of the claim but his article contained six or seven caveats to the effect that the claim might not bear out.”

When the papers were published for all to see, it turned out to be insufficient to demonstrate the claim. Gallo had found the virus in too few of the AIDS patients with actual AIDS symptoms – only 26 out of 72, or 36% – to substantiate his claim. He was unable to demonstrate the presence of the virus in two thirds – 64 per cent – of the AIDS patients sampled.”

Here are the figures as shown in Table 1 of the paper “Frequent Detection and Isolation of Cytopathic Retroviruses (HTLV-III) from Patients with AIDS and Risk for AIDS”, Robert C. Gallo, Syed Z. Salahuddin, Mikulas Popovic, et al, Science, May 4, 1984: 224:500-502:

Group Diagnosed: Number positive for HTLV-III/Number tested/Percent positive

Pre-AIDS?: 18/ 21                85.7% !!!!!

Clinically normal mothers of juvenile AIDS patients: 3/47           5.0%

Juvenile AIDS: 3/ 8             37.5%

Adult AIDS with Kaposi’s sarcoma: 13/ 43              30.2% !!!!!

Adult AIDS with opportunistic infections: 10/ 21           47.6% !!!!!!

Clinically normal homosexual donors: 1/ 22           4.5%

Clinically normal heterosexual donors: 0/115          0%

Or as noted in the article:

As summarized in Table 1, we found HTLV-III in 18 of 21 samples from patients with “pre-AIDS,” from three of four clinically normal mothers of juvenile AIDS patients, 13 of 43 adult AIDS patients with Kaposi’s sarcoma, and 10 of 21 adult AIDS patients with opportunistic infections.”

This result partly veiled the stark failure of the sampling to identify persuasively HIV as a cause of AIDS. For the sum total of AIDS patients with symptoms of AIDS – the groups in bold in the table above – was that in ONLY 26 (3 +13 +10) out of 72 (8 + 43 + 21) cases was the Gallo lab able to show HTLV-III virus detected and isolated.

26 of 72, or 36%, was insufficient to demonstrate that HIV was the plausible cause of the AIDS symptoms or their underlying immune deficiency. If anything, the testing demonstrated that HTL-III was certainly not a plausible cause of AIDS.

Michael Gottlieb, the first to describe AIDS in LA in his 1981 paper, found cytomegalovirus in 100% of his patients, and Montagnier’s Patient One also had cytomegalovirus detected, but Gottlieb wisely concluded that CMV was “opportunistic” and not “causal,” while Montagnier published that “LAV” was “associated with” and not causal, despite his quiet and gracious acceptance of the Nobel a few months ago.

But Gallo concluded: ‘These studies of HTLV-III isolates from patients with AIDS and pre-AIDS and from healthy individuals at risk for AIDS provide strong evidence of a causative involvement of the virus in AIDS.’

Thus contrary to subsequent headlines the Science paper did not state firmly that HTLV-III (later renamed Human Immunodeficiency Virus) was the cause or even a “probable” cause of AIDS, only that there was evidence of a “causative involvement” of the virus in AIDS.

In another paper of the four (Sarngadharan MG, Popovic M,Bruch L, et al. Antibodies Reactive to Human T-Lymphotrophic Retroviruses (HTLV-III) in the Serum of Patients with AIDS. Science 1984:224:506-508), however, the claim was bolder:

The data presented here and in the accompanying reports suggest that HTLV-III is the primary cause of AIDS.’

The low figure of only 36% of AIDS patients with symptoms that had HTLV-III virus present was excused in an accompanying news report in a Science Research News column by Jean Marx as “possibly due to deterioration of the samples.”

When the investigators calculated the percentage, they used the total of all the AIDS samples sent to them, even though some had deteriorated to the point where they were of questionable value for analysis.

The paper itself had noted:

The incidence of virus isolated reported here probably underestimates its true incidence since many tissue specimens were not received or handled under what we now recognize as ideal conditions. This is particularly so for the samples received from late stage AIDS patients.”

That is to say, 26 of the 72 with AIDS tested positive for the virus, 22 of the 47 who did not have AIDS (asymptomatic or pre-AIDS- i.e mild and non-AIDS specific symptoms), suggesting that HTLV-III positivity was a poor guide as to who would develop AIDSsymptoms. Later, this problem was solved by counting as AIDS only those who were HTLV-III positive, a practice that continues to this day, and cements the circular logic that:

HIV-positive result+pneumonia=AIDS—–treatment with antiretrovirals for life and bactrim.

HIV-negative result+pneumonia=pneumonia=treatment for <28 days with bactrim or other antibiotics alone.

To put it even more bluntly, the paper’s landmark finding is the reason why Gallo is forced to leave the number “forty-eight” standing out there in his letter all by itself, not saying 48 out of how many, because in fact he found the virus in blood from 48 individuals out of 119, and 22 of the 48 did not have AIDS, rather than all of them being “individuals who had AIDS.”

It also should be emphasized that “HIV” did not emerge from a Special Virus Program or U.S. government conspiracy for population control or for germ warfare as has been popularized by Dr. Leonard G. Horowitz and others. “HIV” did not crawl out from a monkey or chimp kidney culture used during the manufacture the hepatitis B vaccine or other vaccines, and certainly it didn’t occur during any “close associations” between Africans and non-human primates that Robert Gallo, Max Essex, and the Public Health Service like to imagine for each “slow-virus” these “slow virologists” like to invent. Why are these scenarios unlikely? Because then it would be easy to see a virus in the blood of an AIDS patient, rather than impossible.

The probable cause of “HIV” is….a retroid:

Recent studies in gene research and molecular biology suggest that the so-called specific markers of “HIV” are produced by our own non-specific endogenous DNA sequences called retroelements or “retroids.”

A retroid is a special kind of mobile gene sequence that has been associated with diseases such as multiple sclerosis, and with normal biological functions involving the placenta (8) and umbilical cord, as exhibited by the cord-derived lymphocytes Montagnier used to propagate so-called Patient One’s “LAV-infection” In Vitro, and the accompanying electron micrographs of viral-like particles. It is known that these retroid sequences make cellular proteins that are expressed by normal uninfected (healthy) yeast, insects, and a variety of mammals (9), 50% of healthy dogs (10), “uninfected” rhesus monkeys, chimps, and humans (11).

Retroelements are now known to be important sequences for telomere replication at the tips of normal and cancer cell chromosomes (12). Once claimed by AIDS scientists to be a specific molecular component required for “HIV” replication, retroids and specifically reverse transcriptases (RT) are now seen frequently in market magazines concerning biotechnology stocks (13, 14) in the context of normal, non-pathological situations, despite what AIDS promoters like Robert Gallo, Max Essex, Anthony Fauci, Mark Wainberg, John Moore, Julie Gerberding, the CDC, and the thousands of other AIDS promoters continue to claim about the specificity of RT, p24, and the other mistaken exogenous proteins or glycoproteins used as a surrogate markers used for detecting a supposed exogenous retrovirus, “HIV.”

p24, a protein once thought to be the unique capsid protein that supposedly makes up the proteinaceous shell of “HIV,” is now known to be expressed in the thymus glands of “HIV-negative” children (15). An “HIV” positive result can also occur simply when some infants are exposed to the proteins in cow’s or goat’s milk (16). Two-percent of people will test positive transiently after the flu vaccine (17), or a hepatitis B vaccine can generate a positive “HIV” test result (18), and as mentioned before, normal pregnant women are known to frequently test false positive (19), as do individuals who test under conditions of physiological or extreme emotional stress caused by disease, drugs, oxidation, malnutrition, a fatal diagnosis, and because of dozens of other factors or reasons (20), but paradoxically, not after specific “HIV” sequences or proteins are directly injected into “HIV-negative” “HIV-vaccine recipients after 63 failed “HIV” vaccine trials.

That these endogenous human genetic elements packaged into endosomes exist but yet are ill-defined has been shown again and again to be likely from studies on presumptively and incorrectly named “HERV’s” (Human Endogenous Retro-Viruses) such as the “Phoenix viruses” (presumptively and incorrectly named because nobody has shown that endogenous infectious “retroviruses” cause illness). “HERV’s” (viral-like particles that look like “HIV” virus particles are supposed to look) can be produced by infecting (transfecting in Petri dishes) cells with certain sequences of DNA or RNA (21), which then are replicated and packaged by the cells into virus-like “enveloped” particles that look identical to “HIV.” Modern analyses of the human genome database (which presumably wasn’t derived from anyone infected with “HIV”) have revealed more than 120, 000 full-length retroids containing (once thought to be) specific viral reverse transcriptase transcripts (22).

Although the promoters of “HIV=AIDS” are always saying the “HIV virus’s” reverse transcriptase sequence and other parts of it’s genome are mutating every time a patient dies while on “life saving” anti-retroviral drugs that supposedly target this and other “HIV-specific” gene sequence products, genomic analyses show that these retroid reverse transcriptase elements are among the most stable transcripts that make up these retroids. In other words, amongst gene sequence analysts, some of whom are developing this hypothesis instead of trying to pin the origin of “HIV” on black people’s association with monkeys or apes in Cameroon, or elsewhere, it is the sequence stability rather than the instability or mutability of the reverse transcriptase sequences themselves that make these 120,000 retro-element sequences possible to classify as distinct DNA sequences (22), while at the same time, the AIDS Establishment points to the mutability of these same sequences as the reason why they have failed to find a single stable target in “the AIDS virus,” or why they believe that many viruses ultimately derive from Africans, or black peoples (West Nile was “isolated” from a healthy Ugandan woman who had a cold, Hepatitis B was isolated from a black Australian Aboriginal gentleman, the 6 “HIV-associated cancers,” ultimately came from non-human primates through Africans, etc).

In conclusion, when these facts are added to the political-economic inevitabilities of The Church of Modern Medicine’s Dark-Ages view of Mankind, the formula for disaster becomes especially toxic. This view isn’t that Man is now viewed and treated as if he is inherently sinful, but instead, Man is viewed as inherently physically sick, and in need of medicine, doctors, and medical care from the moment of birth to death.

In the case of “HIV/AIDS,” it all becomes heartbreaking and life-ending. It isn’t just restless leg syndrome, mind you, or the suggestion that our children who are wildly obese begin their statin regimens, or prozac if they are aggressive or moody. And it isn’t that the CDC wants all infants and children to get 20 vaccines or ban our children from entering schools, or charge their parents $500.00 dollars/day until they vaccinate, as what happened to mostly Black parents in Maryland recently (e.g.):

2008, January. Prince George’s County, Upper Marlboro, Maryland (CNN).

A crowd of frustrated parents gathered on a chilly Saturday morning outside Prince George’s County Circuit Court to comply with an order from the school system to have their children vaccinated — or else.

Prince George’s County State’s Attorney Glenn Ivey, whose office began the effort, was at the courthouse to answer questions.

 Judge C. Philip Nichols, who signed the letters threatening parents with jail or fines, said he felt the tactic worked.

 “We got a thousand kids back in school just by sending one letter,” he said.

Nichols ordered parents to come to court Saturday to either immunize the children on the spot, or to provide proof that they already had their shots, according to The Associated Press.

Families who failed to comply could face 10 to 30 days in jail.

You will be convicted of murder, as many have we have tried to help, if you don’t tell folks “your status.” For “HIV-positives,” no less than scarlet letters, or tattoos, have been suggested. These people are not human. They are dehumanized, and this is a difficult thing for people like us (and probably you) to understand. At a minimum, it should be stated clearly that after knowing many of these brave souls during the past 25 years, words are lacking for sure to properly convey the extent of their dehumanization due to the stigma of a positive “HIV” test result.

Let it be expressed this way: “HIV-positives” have no basis for believing they deserve human rights or that they are actually human beings. They can’t travel or enter the U.S. without great hardship. Their doctors won’t even touch their breast cancer biopsies, and will at times run out of the examining room (as did the doctors in the Audrey Serrano case and many others). As the German philosopher Nietzsche once described it, these folks are “undermenchen, ” that really have no business living amongst “ubermenchen” (underman versus overman), and they should be identified, and then “weeded out.”

The pure and proud institutionalized racisim and homophobia that emerges from such a belief system is no different than what the Nazi’s achieved: “infected” or “defective people” eventually should be sterilized or weeded out of the gene pool completely through various and vigorous eugenics pogroms.

Certainly, they shouldn’t be allowed to reproduce or pass on their “infections” or defects. These individuals will eventually include, the mentally and emotionally “different,” as well as homosexuals, criminals, Africans, Hispanics, Jews, Muslims, Zorastrians, Amish, “HIV-positives,” perhaps pianists someday, and other groups that may be deemed “high risk.”

Even when there is no illness, but simply a molecular signature or marker

detected by The Church of Modern Medicine, these people should not be allowed to reproduce or pass their “infection” to others without facing criminal charges and incarceration. The people themselves have no worth, as you see them come into your examination office. It’s easy to convince them of this logic, and continuously reinforce it, when mandated by The State. Have they been “compliant” you ask? Do they “know their status?” Do you know your status?

Meanwhile, even while experiencing perfect health but now carrying the State Government-reportable “HIV-infection,” “the patient” begins the long struggle with paralyzing feelings of isolation and violence against them, that they feel is directed at them. Anger, pain, and great paranoia are commonly described. They have a high suicide rate as they internalize these feelings, because who would wish to live under these circumstances?

If it is a child of a parent who “is infected” (despite a 60% sero-reversion rate after 9-18 months after birth), the parents can be held for criminal negligence unless they submit to having the children drugged to the extent that neurological demise develops, or drug-induced diseases occur, as in the case of the more than 300 orphans drugged through g-tubes with 7 black box label drugs at New York’s Incarnation Children’s Center (ICC), and elsewhere (Chicago’s Northwestern Hospital), or, when many of them are taken away from the natural parents or legal guardians to be made compliant with the drug regimens, after which only about 80 deaths were recently admitted in one trial (at ICC).

Mothers and infants who are “HIV-positive” are dissuaded from bonding during the time of birth, and parents are brow beaten and threatened by The State in many cases to force their infant to imbibe black-box-label drugs through forced administration or through surgically-implanted g-tubes with drugs that have been shown to be able to induce brain abnormalities, liver failure, stunted growth, and death. Breast feeding is forbotten! To do otherwise is, irresponsible.

Why not instead, I ask, predicate these global policies upon proper science and honest empirical assessments of truly promising programs as advocated by Doctors Without Borders, as opposed to the pharma-directed and iatrogenic perversions of science that have and are still occurring among The Church of Modern Medicine, politicians, the media, and a highly dishonest and self-promoting bunch of pharma and foundation-backed scientists like John Moore of Cornell’s Medical college? One example of such a promising program should suffice, and also, should guide the future Global AIDS Coordinator, whomever this person will be now that Dybul has been “relieved.” Here are a few segments from a story that recently appeared on 60 minutes:


Oct. 21, 2007. Doctors Without Borders Briefing Paper: Food Is Not Enough: African children need food, not drugs (See the video free at:


Plumpynut is cheap, nutritious and needs no refrigeration. It is saving starving children in the developing world and could save more … if there were more of it.

You’ve probably never heard a good news story about malnutrition, but you’re about to. Every year, malnutrition kills five million children — that’s one child every six seconds. But now, the Nobel Prize-winning relief group “Doctors Without Borders” says it finally has something that can save millions of these children.

It’s cheap, easy to make and even easier to use. What is this miraculous cure? As CNN’s Anderson Cooper reports, it’s a ready-to-eat, vitamin-enriched concoction called “Plumpynut,” an unusual name for a food that may just be the most important advance ever to cure and prevent malnutrition.

“It’s a revolution in nutritional affairs,” says Dr. Milton Tectonidis, the chief nutritionist for Doctors Without Borders.

“Now we have something. It is like an essential medicine. In three weeks, we can cure a kid that is looked like they’re half dead. We can cure them just like an antibiotic. It’s just, boom! It’s a spectacular response,” Dr. Tectonidis says.

“It’s the equivalent of penicillin, you’re saying?” Cooper asks.

“For these kids, for sure,” the doctor says.

No kids need it more than a group of children 60 Minutes saw in Niger, a desperately poor country in West Africa, where child malnutrition is so widespread that most mothers have watched at least one of their children die.

Why are so many kids dying? Because they can’t get the milk, vitamins and minerals their young bodies need. Mothers in these villages can’t produce enough milk themselves and can’t afford to buy it. Even if they could, they can’t store it — there’s no electricity, so no refrigeration. Powdered milk is useless because most villagers don’t have clean water. Plumpynut was designed to overcome all these obstacles.

Plumpynut is a remarkably simple concoction: it is basically made of peanut butter, powdered milk, powdered sugar, and enriched with vitamins and minerals. It tastes like a peanut butter paste. It is very sweet, and because of that kids cannot get enough of it.

The formula was developed by a nutritionist. It doesn’t need refrigeration, water, or cooking; mothers simply squeeze out the paste. Many children can even feed themselves. Each serving is the equivalent of a glass of milk and a multivitamin.

Niger has become Plumpynut’s proving ground. A daily dose costs about $1; small factories mix it here and in three other African countries. Tectonidis says other companies could make similar products wherever children need them.

“There’s many countries in Africa now saying, ‘We want a factory. We want a factory.’ Well let’s give it to them,” he says. “We just have to focus on these areas. We don’t have to feed the whole world. We have to go for the jugular. Where are they dying? Where are they wasted? That’s where we have to intervene. If you feed them well until they’re two or three years old it’s won. They’re healthy, they can get a healthy life. If you miss that window, it’s finished.”

Normally a children’s hospital 60 Minutes visited would have more patients than beds. But now, thanks to Plumpynut, it has empty beds. Dr. Susan Shepherd, a pediatrician from Butte, Mont., runs Doctors Without Borders in Niger.

She says children that would have been hospitalized in the past can now be treated at home. “The reason we can do that is because we can give children Plumpynut here in the ambulatory center, and they take a week’s ration home. Moms treat their children at home and come back every week for a weight check,” Dr. Shepherd explains.

CBS) If Plumpynut is the answer, how come kids are still dying?

“The answer is getting to kids earlier,” Shepherd says. “Once children are as sick as she is, Plumpynut is not gonna save her.”

Rashida was buried in a nearby cemetery. The grave digger, Salifu Ibrahim, told 60 Minutes he used to dig graves for about seven children a day, but now, on most days, he digs only one.

Asked why he thinks fewer children are dying, Ibrahim says, “It is God’s will.”

God’s will and Plumpynut.

Two years ago this region had the highest malnutrition rate in Niger. But now, after widespread use of the Plumpynut, it has the lowest. Dr. Shepherd told Cooper they’ll be able to treat more than 120,000 kids this year, up from just 10,000 children three years ago.

It’s hard to imagine a less industrialized country than Niger. On a list of 177 developing countries, the United Nations ranked Niger dead last — least developed. More than 70 percent of the people don’t know how to read. Most work in the fields and earn less than a dollar a day. Nomadic goat herders still roam this land — their children and their kids travel by camel. Goats seem to be the main garbage disposal, but clearly the goats are falling behind. You can still spot a skinny guard dog, but we were told all the cats have been cooked.

In the countryside, where 85 percent of people live, girls start marrying as young as 11 years old. By the age of 15 most are wed, and by 16 most have already become mothers. The average woman here will give birth at least eight times in her lifetime. But largely because of malnutrition, one in five of their children will die before they reach the age of five. Of those who survive, half will have stunted growth and never reach full adult height.

But now, with Plumpynut, more children are surviving and thriving.

Doctors Without Borders is asking for more of this type of food. Their success in Niger proves, they say, that fortified ready-to-eat products, like Plumpynut, save children’s lives. Dr. Tectonidis says if the United States and the European Union were willing to spend part of their food aid on this, more companies will start making it.

“Even by taking a miniscule proportion of the global food aid budget, they will have a huge impact, huge impact!” Tectonidis says. “We’re not even asking for billions. It will solve so much of the underlying useless death. So we gotta do that now.”

“It’s useless death,” Cooper remarks.

“Wasted life. Just totally wasted life for nothing. Because they don’t have this product, little a bit of peanut butter with vitamins,” Tectonidis says. “What a waste.”


1. Josephson et al. Investigation of Atypical Western Blot (immunoblot)

Reactivities Involving Core Proteins of Human Immunodifficiency Virus Type

I. Journal of Clinical Microbiology, May 1989, p, 932-937 , 1989.

2. Papadopulos-Eleopulos et al; Is a Positive Western Blot Proof of HIV

Infection ? Bio/Technology 1993; 11:696-707; Papadopulos-Eleopulos et al..

HIV Antibodies: Further Questions and a Plea for Clarification. Curr Med

Res Opin 1997; 13:627-634; Johnson C. Factors Known to Cause

False-Positive HIV Antibody Test Results; Zenger’s San Diego, California,

September 1996: 8-9; Johnson C.JOHNSON C. Whose Antibodies Are They

Anyway? Continuum (London), September/October 1996; 4(3):4-5; Hodgekinson

N. Science Fails the “AIDS Test”. In: AIDS: The Failure of Contemporary

Science. How a Virus that Never Was Deceived the World. London: Fourth

Estate, 1996: 232-262.

3. Rodriquez B, Sethi AK, Cheruvu VK, et al. Predictive value of plasma

HIV RNA level on rate of CD4 T-cell decline in untreated HIV infection.

JAMA 296(12):1498-506, 2006.

4. Fauci, A.S. Mechanisms of Corticosteroid Action on lymphocyte

Subpopulations I. Redistribution of circulating T and B lymphocytes to the

bone marrow. Immunology 28: 669-679. 1975.

5. Fauci, A.S., Dale, D.C., and Balow, J.E. Glucocorticosteroid therapy:

Mechanisms of Action and Clinical Considerations. Annals of Internal

Medicine 84: 304-15, 1976.

6. Noireau F. HIV transmission from monkey to man. Lancet (i): 1498-1499,


7. Green J., Miller D. AIDS The story of a disease, p66. Grafton Books,

London 1986

8. Retroelements and the human genome: new perspectives on an old

relation. Suppl 2:14572-9. Aug 13, Epub 2004.

9. Varmus H. Reverse transcription Sci. Am. 257:48-54, 1987.

10. Strandstrom et al., Studies with canine sera that contain antibodies

which recognize human immunodeficiency virus structural proteins Cancer

Res 1990 Sep 1;50 17 Suppl:5628S-5630S.

11. Horwitz MS, Boyce-Jacino MT, Faras AJ. Novel human endogenous

sequences related to human immunodeficiency virus type 1. J Virol. Apr;66

(4):2170-9, 1992.

12. Ghori A. 1; Usselmann B. 2; Odogwu S. 1; Fraser I. 1; Morris A. 2

Colorectal Disease,, vol. 2, no. 2, pp. 106-112, 2000.

13. Pachecz M. No need to be phased. Shares, Vol. 6, 28-32, 2001.

14. Papadopulos-Eleopulos E, Turner VF, Papadimitriou J, Page B, Causer

D, Alfonso H, Mhlongo S, Miller T, Maniotis A, Fiala C. A critique of the

Montagnier evidence for the HIV/AIDS hypothesis. Med Hypotheses

63(4):597-601, 2004.

15. Dura WT, Wozniewicz BM. Expression of antigens homologous to human

retrovirus molecules in normal and severely atrophic thymus. Thymus 22

(4):245-54, 1994.

16. Willman et al., Heterophile Antibodies to Bovine and Caprine Proteins

Causing False-Positive Human Immunodeficiency Virus Type 1 and Other.

Enzyme-Linked Immunosorbent Assay Results. Clinical and Diagnostic

Laboratory Immunology, p. 615-616, Vol. 6, No. 4, July 1999.

17. Simonsen L, Buffington J, Shapiro CN, et al. Multiple false reactions

in viral antibody screening assays after influenza vaccination. Am J

Epidemiol 141:1089-1096,1995.


18. Lee, D, Eby W, Molinaro, G.. HIV false positivity after Hepatitis B

vaccination. Lancet 339: 1060, 1992.

19. Doran TI et al. False-Positive and Indeterminate Human

Immunodeficiency Virus Test Results in Pregnant Women. Arch Fam Med.

Sep/Oct; 9: 924-9, 2000.

20. Christine Johnson. Whose antibodies are they anyway? Factors known to

cause false postive HIV test results. Continuum. Sept/Oct. 1996.


21. Marie Dewannieux, Francis Harper, Aurelien Richaud, Claire Letzelter,

David Ribet, Gerard Pierron, and Thierry Heidmann. Identification of an

infectious progenitor for the multiple-copy HERV-K human endogenous

retroelements. Genome Res. Oct. 31, 2006.Bannert N, Kurth R. Proc Natl

Acad Sci U S A. 2004 Oct 5;101.

22. McClure MA, Richardson HS, Clinton RA, Hepp CM, Crowther BA, Donaldson EF. Automated characterization of potentially active retroid agents in the human genome. Genomics. Apr;85(4):512-23, 2005.


Factors Known to Cause False Positive HIV Antibody Test Results:

By Christine Johnson, Continuum Sept./Oct. 1996


The AIDS establishment has managed to convince many people that the HIV antibody tests (ELISA, IFA and Western blot) are “99.5% accurate”. In this article Christine Johnson from HEAL Los Angeles, lists conditions documented in the scientific literature known to cause positives on these tests, and gives her references.

It is expected that this list will generate much discussion and dissension. For the time being, a few clarifications should be made at the outset.

Just because something is on the list doesn’t mean that it will definitely, or even probably, cause a false-positive. It depends on what antibodies the individual carries; as well as the characteristics of each particular test kit.

For instance, some, but not all, people who have had blood transfusions, prior pregnancies or an organ transplant will make HLA antibodies. And some, but not all, test kits (both ELISA and Western blot) will be contaminated with HLA antigens to which these antibodies can react. Only if these two conditions coincide might you get a false-positive due to HLA cross-reactivity.

Some things are more likely than others to cause false-positives. And some things that we aren’t aware of yet, but which may be documented in the future, may cause false-positives. Some of the factors on the list have been documented only for ELISA, some for both ELISA and Western blot (WB).

Some people may be eager to argue that if a factor is only known to cause false-positives on ELISA, this problem won’t be carried over to the WB, so everything should be OK. But remember, a WB is positive by virtue of accumulating enough individual positive bands to add up to the total required by whatever criteria you use to interpret it (39) So the more exposures a person has had to foreign antigens, proteins and infectious agents, the more various antibodies he or she will have in their system, and the more likely it is that there will be several cross-reacting antibodies, enough to make the WB positive.

It is to be noted that all AIDS risk groups (and Africans as well), but not the general US or Western European population, have this problem in common: they have been exposed to a plethora of foreign antigens and proteins. This is why people in the AIDS “risk groups” tend to have positive WBs (i.e., to be considered “HIV-infected”) and people in the general population don’t. However, even people in the low-risk populations may have false-positive Western blots for poorly understood reasons.(47)

Since false-positives to every single HIV protein have been documented (36), how do you know the positive WB bands represent the various proteins to HIV, or just a collection of false-positive bands reacting to several different non-HIV antibodies?


Factors Known to Cause False-Positive HIV Antibody Test Results:


* Anti-carbohydrate antibodies (52, 19, 13)

* Naturally-occurring antibodies (5, 19)

* Passive immunization: receipt of gamma globulin or immune globulin (as prophylaxis against infection which contains antibodies)(18, 26, 60, 4, 22, 42, 43, 13)

* Leprosy (2, 25)

* Tuberculosis (25)

* Mycobacterium avium (25)

* Systemic lupus erythematosus (15, 23)

* Renal (kidney) failure (48, 23, 13)

* Hemodialysis/renal failure (56, 16, 41, 10, 49)

* Alpha interferon therapy in hemodialysis patients (54)

* Flu (36)

* Flu vaccination (30, 11, 3, 20, 13, 43)

* Herpes simplex I (27)

* Herpes simplex II (11)

* Upper respiratory tract infection (cold or flu)(11)

* Recent viral infection or exposure to viral vaccines (11)

* Pregnancy in multiparous women (58, 53, 13, 43, 36)

* Malaria (6, 12)

* High levels of circulating immune complexes (6, 33)

* Hypergammaglobulinemia (high levels of antibodies) (40, 33)

* False positives on other tests, including RPR (rapid plasma reagent) test for syphilis (17, 48, 33, 10, 49)

* Rheumatoid arthritis (36)

* Hepatitis B vaccination (28, 21, 40, 43)

* Tetanus vaccination (40)

* Organ transplantation (1, 36)

* Renal transplantation (35, 9, 48, 13, 56)

* Anti-lymphocyte antibodies (56, 31)

* Anti-collagen antibodies (found in gay men, haemophiliacs, Africans of both sexes and people with leprosy)(31)

* Serum-positive for rheumatoid factor, antinuclear antibody (both found in rheumatoid arthritis and other auto- antibodies)(14, 62, 53)

* Autoimmune diseases (44, 29, 10, 40, 49, 43): Systemic lupus erythematosus, scleroderma, connective tissue disease, dermatomyositis

* Acute viral infections, DNA viral infections (59, 48, 43, 53, 40, 13)

* Malignant neoplasms (cancers)(40)

* Alcoholic hepatitis/alcoholic liver disease (32, 48, 40,10,13, 49, 43, 53)

* Primary sclerosing cholangitis (48, 53)

* Hepatitis (54)

* “Sticky” blood (in Africans) (38, 34, 40)

* Antibodies with a high affinity for polystyrene (used in the test kits)(62, 40, 3)

* Blood transfusions, multiple blood transfusions (63, 36,13, 49, 43, 41)

* Multiple myeloma (10, 43, 53)

* HLA antibodies (to Class I and II leukocyte antigens)(7, 46, 63, 48, 10, 13, 49, 43, 53)

* Anti-smooth muscle antibody (48)

* Anti-parietal cell antibody (48)

* Anti-hepatitis A IgM (antibody)(48)

* Anti-Hbc IgM (48)

* Administration of human immunoglobulin preparations pooled before 1985 (10)

* Haemophilia (10, 49)

* Haematologic malignant disorders/lymphoma (43, 53, 9, 48, 13)

* Primary biliary cirrhosis (43, 53, 13, 48)

* Stevens-Johnson syndrome9, (48, 13)

* Q-fever with associated hepatitis (61)

* Heat-treated specimens (51, 57, 24, 49, 48)

* Lipemic serum (blood with high levels of fat or lipids)(49)

* Haemolyzed serum (blood where haemoglobin is separated from the red cells)(49)

* Hyperbilirubinemia (10, 13)

* Globulins produced during polyclonal gammopathies (which are seen in AIDS risk groups)(10, 13, 48)

* Healthy individuals as a result of poorly-understood cross-reactions (10)

* Normal human ribonucleoproteins (48,13)

* Other retroviruses (8, 55, 14, 48, 13)

* Anti-mitochondrial antibodies (48, 13)

* Anti-nuclear antibodies (48, 13, 53)

* Anti-microsomal antibodies (34)

* T-cell leukocyte antigen antibodies (48, 13)

* Proteins on the filter paper (13)

* Epstein-Barr virus (37)

* Visceral leishmaniasis (45)

* Receptive anal sex (39, 64)


References for Factors Known to Cause False Positive HIV Antibody Test Results:


1. Agbalika F, Ferchal F, Garnier J-P, et al. 1992. False-positive antigens related to emergence of a 25-30 kD protein detected in organ recipients. AIDS. 6:959-962.

2. Andrade V, Avelleira JC, Marques A, et al. 1991. Leprosy as a cause of false-positive results in serological assays for the detection of antibodies to HIV-1. Intl. J. Leprosy. 59:125.

3. Arnold NL, Slade RA, Jones MM, et al. 1994. Donor follow up of influenza vaccine-related multiple viral enzyme immunoassay reactivity. Vox Sanguinis. 67:191.

4. Ascher D, Roberts C. 1993. Determination of the etiology of seroreversals in HIV testing by antibody fingerprinting. AIDS. 6:241.

5. Barbacid M, Bolgnesi D, Aaronson S. 1980. Humans have antibodies capable of recognizing oncoviral glycoproteins: Demonstration that these antibodies are formed in response to cellular modification of glycoproteins rather than as consequence of exposure to virus. Proc. Natl. Acad. Sci. 77:1617-1621.

6. Biggar R, Melbye M, Sarin P, et al. 1985. ELISA HTLV retrovirus antibody reactivity associated with malaria and immune complexes in healthy Africans. Lancet. ii:520-543.

7. Blanton M, Balakrishnan K, Dumaswala U, et al. 1987. HLA antibodies in blood donors with reactive screening tests for antibody to the immunodeficiency virus. Transfusion. 27(1):118.

8. Blomberg J, Vincic E, Jonsson C, et al. 1990. Identification of regions of HIV-1 p24 reactive with sera which give “indeterminate” results in electrophoretic immunoblots with the help of long synthetic peptides. AIDS Res. Hum. Retro. 6:1363.

9. Burkhardt U, Mertens T, Eggers H. 1987. Comparison of two commercially available anti-HIV ELISA’s: Abbott HTLV-III ELA and DuPont HTLV-III ELISA. J. Med. Vir. 23:217.

10. Bylund D, Ziegner U, Hooper D. 1992 Review of testing for human immunodeficiency virus. Clin. Lab. Med. 12:305-333.

11. Challakere K, Rapaport M. 1993. False-positive human immunodeficiency virus type 1 ELISA results in low-risk subjects. West. J. Med. 159(2):214-215.

12. Charmot G, Simon F. 1990. HIV infection and malaria. Revue du practicien. 40:2141.

13. Cordes R, Ryan M. 1995. Pitfalls in HIV testing. Postgraduate Medicine. 98:177.

14. Dock N, Lamberson H, O’Brien T, et al. 1988. Evaluation of atypical human immunodeficiency virus immunoblot reactivity in blood donors. Transfusion. 28:142.

15. Esteva M, Blasini A, Ogly D, et al. 1992. False positive results for antibody to HIV in two men with systemic lupus erythematosus. Ann. Rheum. Dis. 51:1071-1073.

16. Fassbinder W, Kuhni P, Neumayer H. et al. 1986. Prevalence of antibodies against LAV/HTLV-III [HIV] in patients with terminal renal insufficiency treated with hemodialysis and following renal transplantation. Deutsche Medizinische Wochenschrift. 111:1087.

17. Fleming D, Cochi S, Steece R. et al. 1987. Acquired immunodeficiency syndrome in low-incidence areas. JAMA. 258(6):785.

18. Gill MJ, Rachlis A, Anand C. 1991. Five cases of erroneously diagnosed HIV infection. Can. Med. Asso. J. 145(12):1593.

19. Healey D, Bolton W. 1993. Apparent HIV-1 glycoprotein reactivity on Western blot in uninfected blood donors. AIDS. 7:655-658.

20. Hisa J. 1993. False-positive ELISA for human immunodeficiency virus after influenza vaccination. JID. 167:989.

21. Isaacman S. 1989. Positive HIV antibody test results after treatment with hepatitis B immune globulin. JAMA. 262:209.

22. Jackson G, Rubenis M, Knigge M, et al. 1988. Passive immunoneutralisation of human immunodeficiency virus in patients with advanced AIDS. Lancet, Sept. 17:647.

23. Jindal R, Solomon M, Burrows L. 1993. False positive tests for HIV in a woman with lupus and renal failure. NEJM. 328:1281-1282.

24. Jungkind D, DiRenzo S, Young S. 1986. Effect of using heat-inactivated serum with the Abbott human T-cell lymphotropic virus type III [HIV] antibody test. J. Clin. Micro. 23:381.

25. Kashala O, Marlink R, Ilunga M. et al. 1994. Infection with human immunodeficiency virus type 1 (HIV-1) and human T-cell lymphotropic viruses among leprosy patients and contacts: correlation between HIV-1 cross-reactivity and antibodies to lipoarabionomanna. J. Infect. Dis. 169:296-304.

26. Lai-Goldman M, McBride J, Howanitz P, et al. 1987. Presence of HTLV-III [HIV] antibodies in immune serum globulin preparations. Am. J. Clin. Path. 87:635.

27. Langedijk J, Vos W, Doornum G, et al. 1992. Identification of cross-reactive epitopes recognized by HIV-1 false-positive sera. AIDS. 6:1547-1548.

28. Lee D, Eby W, Molinaro G. 1992. HIV false positivity after hepatitis B vaccination. Lancet. 339:1060.

29. Leo-Amador G, Ramirez-Rodriguez J, Galvan-Villegas F, et al. 1990. Antibodies against human immunodeficiency virus in generalized lupus erythematosus. Salud Publica de Mexico. 32:15.

30. Mackenzie W, Davis J, Peterson D. et al. 1992. Multiple false-positive serologic tests for HIV, HTLV-1 and hepatitis C following influenza vaccination, 1991. JAMA. 268:1015-1017.

31. Mathe G. 1992. Is the AIDS virus responsible for the disease? Biomed & Pharmacother. 46:1-2.

32. Mendenhall C, Roselle G, Grossman C, et al. 1986. False-positive tests for HTLV-III [HIV] antibodies in alcoholic patients with hepatitis. NEJM. 314:921.

33. Moore J, Cone E, Alexander S. 1986. HTLV-III [HIV] seropositivity in 1971-1972 parenteral drug abusers – a case of false-positives or evidence of viral exposure? NEJM. 314:1387-1388.

34. Mortimer P, Mortimer J, Parry J. 1985. Which anti-HTLV-III/LAV [HIV] assays for screening and comfirmatory testing? Lancet. Oct. 19, p873.

35. Neale T, Dagger J, Fong R, et al. 1985. False-positive anti-HTLV-III [HIV] serology. New Zealand Med. J. October 23.

36. Ng V. 1991. Serological diagnosis with recombinant peptides/proteins. Clin. Chem. 37:1667-1668.

37. Ozanne G, Fauvel M. 1988. Perfomance and reliability of five commercial enzyme-linked immunosorbent assay kits in screening for anti-human immunodeficiency virus antibody in high-risk subjects. J. Clin. Micro. 26:1496.

38. Papadopulos-Eleopulos E. 1988. Reappraisal of AIDS – Is the oxidation induced by the risk factors the primary cause? Med. Hypo. 25:151.

39. Papadopulos-Eleopulos E, Turner V, and Papadimitriou J. 1993. Is a positive Western blot proof of HIV infection? Bio/Technology. June 11:696-707.

40. Pearlman ES, Ballas SK. 1994. False-positive human immunodeficiency virus screening test related to rabies vaccination. Arch. Pathol. Lab. Med. 118-805.

41. Peternan T, Lang G, Mikos N, et al. Hemodialysis/renal failure. 1986. JAMA. 255:2324.

42. Piszkewicz D. 1987. HTLV-III [HIV] antibodies after immune globulin. JAMA. 257:316.

43. Profitt MR, Yen-Lieberman B. 1993. Laboratory diagnosis of human immunodeficiency virus infection. Inf. Dis. Clin. North Am. 7:203.

44. Ranki A, Kurki P, Reipponen S, et al. 1992. Antibodies to retroviral proteins in autoimmune connective tissue disease. Arthritis and Rheumatism. 35:1483.

45. Ribeiro T, Brites C, Moreira E, et al. 1993. Serologic validation of HIV infection in a tropical area. JAIDS. 6:319.

46. Sayers M, Beatty P, Hansen J. 1986. HLA antibodies as a cause of false-positive reactions in screening enzyme immunoassays for antibodies to human T-lymphotropic virus type III [HIV]. Transfusion. 26(1):114.

47. Sayre KR, Dodd RY, Tegtmeier G, et al. 1996. False-positive human immunodeficiency virus type 1 Western blot tests in non-infected blood donors. Transfusion. 36:45.

48. Schleupner CJ. Detection of HIV-1 infection. In: (Mandell GI, Douglas RG, Bennett JE, eds.) Principles and Practice of Infectious Diseases, 3rd ed. New York: Churchill Livingstone, 1990:1092.

49. Schochetman G, George J. 1992. Serologic tests for the detection of human immunodeficiency virus infection. In AIDS Testing Methodology and Management Issues, Springer-Verlag, New York.

50. Simonsen L, Buffington J, Shapiro C, et al. 1995. Multiple false reactions in viral antibody screening assays after influenza vaccination. Am. J. Epidem. 141-1089.

51. Smith D, Dewhurst S, Shepherd S, et al. 1987. False-positive enzyme-linked immunosorbent assay reactions for antibody to human immunodeficiency virus in a population of midwestern patients with congenital bleeding disorders. Transfusion. 127:112.

52. Snyder H, Fleissner E. 1980. Specificity of human antibodies to oncovirus glycoproteins; Recognition of antigen by natural antibodies directed against carbohydrate structures. Proc. Natl. Acad. Sci. 77:1622-1626.

53. Steckelberg JM, Cockerill F. 1988. Serologic testing for human immunodeficiency virus antibodies. Mayo Clin. Proc. 63:373.

54. Sungar C, Akpolat T, Ozkuyumcu C, et al. Alpha interferon therapy in hemodialysis patients. Nephron. 67:251.

55. Tribe D, Reed D, Lindell P, et al. 1988. Antibodies reactive with human immunodeficiency virus gag-coated antigens (gag reactive only) are a major cause of enzyme-linked immunosorbent assay reactivity in a bood donor population. J. Clin. Micro. April:641.

56. Ujhelyi E, Fust G, Illei G, et al. 1989. Different types of false positive anti-HIV reactions in patients on hemodialysis. Immun. Let. 22:35-40.

57. Van Beers D, Duys M, Maes M, et al. Heat inactivation of serum may interfere with tests for antibodies to LAV/HTLV-III [HIV]. J. Vir. Meth. 12:329.

58. Voevodin A. 1992. HIV screening in Russia. Lancet. 339:1548.

59. Weber B, Moshtaghi-Borojeni M, Brunner M, et al. 1995. Evaluation of the reliability of six current anti-HIV-1/HIV-2 enzyme immunoassays. J. Vir. Meth. 55:97.

60. Wood C, Williams A, McNamara J, et al. 1986. Antibody against the human immunodeficiency virus in commercial intravenous gammaglobulin preparations. Ann. Int. Med. 105:536.

61. Yale S, Degroen P, Tooson J, et al. 1994. Unusual aspects of acute Q fever-associated hepatitis. Mayo Clin. Proc. 69:769.

62. Yoshida T, Matsui T, Kobayashi M, et al. 1987. Evaluation of passive particle agglutination test for antibody to human immunodeficiency virus. J. Clin. Micro. Aug:1433.

63. Yu S, Fong C, Landry M, et al. 1989. A false positive HIV antibody reaction due to transfusion-induced HLA-DR4 sensitization. NEJM.320:1495.

64. National Institue of Justice, AIDS Bulletin. Oct. 1988.


Additional Human immunodeficiency virus false positive reports between 1996-2011 compiled by Christine Johnson:

-Abdulla EM (2005) False-positive HIV test. J Pak Med Assoc 55: 130.

-Agarwal D, Agrawal NR (2008) False positive HIV-1 DNA PCR in infancy. Indian Pediatr 45: 245-246.

-Araujo PR, et al. (2009) Rubella vaccination and transitory false-positive test results for human immunodeficiency virus Type 1 in blood donors. Transfusion 49: 2516-2517.

-Barbouche RM, et al. (1999) False-positive HIV-1 p24 antigenemia with unusual pattern of neutralization. Arch Inst Pasteur Tunis 76: 11-12.

-Barreiro G, et al. (2007) From docking false-positive to active anti-HIV agent. J Med Chem 50: 5324-5329.

-Bhattacharya R, et al. (2008) When good news is bad news: psychological impact of false positive diagnosis of HIV. AIDS Care 20: 560-564.

-Bouillon M, et al. (2002) Reduced frequency of blood donors with false-positive HIV-1 and -2 antibody EIA reactivity after elution of low-affinity nonspecific natural antibodies. Transfusion 42: 1046-1052.

-Bronze MS, et al. (1998) False-positive enzyme immunoassay for human immunodeficiency virus due to acute cytomegalovirus infection. Clin Infect Dis 27: 221-222.

-Cable RG, et al. (1997) Interpretation of false-positive human immunodeficiency virus type 1 western blot assays in blood donors. Transfusion 37: 670.

-Chintu C, et al. (1997) False-positive self-reports of HIV infection. Lancet 349: 650.

-Chintu C, et al. (1998) False-positive self-reports of HIV-1 infection. Lancet 351: 376.

-del Rio C (2006) Report from the 13th retrovirus conference. More on false-positive results in rapid HIV testing. AIDS Clin Care 18: 39.

-Doran TI, Parra E (2000) False-positive and indeterminate human immunodeficiency virus test results in pregnant women. Arch Fam Med 9: 924-929.

-Erickson CP, et al. (2006) Influenza vaccination and false positive HIV results. N Engl J Med 354: 1422-1423.

-Everett DB, et al. (2010) Association of schistosomiasis with false-positive HIV test results in an African adolescent population. J Clin Microbiol 48: 1570-1577.

-Facente SN, et al. (2009) False positive rate of rapid oral fluid HIV tests increases as kits near expiration date. PLoS ONE 4: e8217.

-Gasasira AF, et al. (2006) False-positive results of enzyme immunoassays for human immunodeficiency virus in patients with uncomplicated malaria. J Clin Microbiol 44: 3021-3024.

-Guinn D (2007) HIV screening and false-positive results. Jama 297: 947; author reply 948.

-Haddow LJ, Robinson AJ (2005) A case of a false positive result on a home HIV test kit obtained on the internet. Sex Transm Infect 81: 359.

-Havlichek DH, Jr., Hage-Korban E (1999) False-positive HIV diagnosis by HIV-1 plasma viral load testing. Ann Intern Med 131: 794.

-Henderson S, et al. (2002) False-positive human immunodeficiency virus seroconversion is not common following rabies vaccination. Clin Diagn Lab Immunol 9: 942-943.

-Hernandez-Aguado I, et al. (1998) False-positive tests for syphilis associated with human immunodeficiency virus and hepatitis B virus infection among intravenous drug abusers. Valencian Study Group on HIV Epidemiology. Eur J Clin Microbiol Infect Dis 17: 784-787.

-Imai M, et al. (1999) [Kinetic analysis of anti-HIV titer in early stage of HIV infection–a new testing strategy to differentiate early HIV infection individuals from false positive cases]. Kansenshogaku Zasshi 73: 1183-1186.

-Jafa K, et al. (2007) Investigation of false positive results with an oral fluid rapid HIV-1/2 antibody test. PLoS ONE 2: e185.

-Joyanes P, et al. (1998) The association of false-positive rapid plasma reagin results and HIV infection. Sex Transm Dis 25: 569-571.

-Kakaiya R, et al. (2011) False-positive nucleic acid test results for human immunodeficiency virus RNA and hepatitis C virus RNA: an underappreciated problem. Transfusion 51: 225-226.

-Kim S, et al. (2010) False-positive rate of a “fourth-generation” HIV antigen/antibody combination assay in an area of low HIV prevalence. Clin Vaccine Immunol 17: 1642-1644.

-Kleinman S, et al. (1998) False-positive HIV-1 test results in a low-risk screening setting of voluntary blood donation. Retrovirus Epidemiology Donor Study. Jama 280: 1080-1085.

-Liu P, et al. (2008) The false-positive and false-negative predictive value of HIV antibody test in the Chinese population. J Med Screen 15: 72-75.

-Magee LA, et al. (1999) False-positive results in antenatal HIV screening. Cmaj 160: 1285.

-Marinovich A, et al. (2006) False-positive result from a Bayer Versant human immunodeficiency virus type 1 branched-DNA viral load assay, with a possible role for light leakage after inadequate maintenance of the analyzer. J Clin Microbiol 44: 4288-4289.

-Mylonakis E, et al. (2000) Report of a false-positive HIV test result and the potential use of additional tests in establishing HIV serostatus. Arch Intern Med 160: 2386-2388.

-Nastouli E, et al. (2007) False-positive HIV antibody results with ultrasensitive serological assays in uninfected infants born to mothers with HIV. Aids 21: 1222-1223.

-Parra-Pineros JE, et al. (2004) False-positive human immunodeficiency virus test and Trypanosoma cruzi infection in eastern Colombia. South Med J 97: 423-424.

-Rapp C, et al. (2005) [Malaria and false positive serology for HIV]. Med Trop (Mars) 65: 397-398.

-Rothman RE, Kalish B (2009) Update on emerging infections: news from the Centers for Disease Control and Prevention. False-positive oral fluid rapid HIV tests–New York City, 2005-2008. Ann Emerg Med 53: 151-154; discussion 154-156.

-Salgado CD, et al. (2002) Low rate of false-positive results with use of a rapid HIV test. Infect Control Hosp Epidemiol 23: 335-337.

-Salinas A, et al. (2007) Refrain from telling bad news: patients with leishmaniasis can have false-positive HIV test results. Clin Infect Dis 45: 139-140.

-Sayre KR, et al. (1996) False-positive human immunodeficiency virus type 1 western blot tests in noninfected blood donors. Transfusion 36: 45-52.

-Schwartz DH, et al. (1997) Extensive evaluation of a seronegative participant in an HIV-1 vaccine trial as a result of false-positive PCR. Lancet 350: 256-259.

-Seme K, et al. (2006) False-positive result of a confirmatory human immunodeficiency virus line immuno assay in an apparently healthy individual–a case report. Coll Antropol 30 Suppl 2: 43-46.

-Sheikh AA, et al. (2004) High frequency of false positive results in HIV screening in blood banks. J Ayub Med Coll Abbottabad 16: 28-31.

-Sheikh AA, et al. (2006) High frequency of false positive results in HIV screening in blood banks. J Pak Med Assoc 56: S72-75.

-Shima-Sano T, et al. (2010) A human immunodeficiency virus screening algorithm to address the high rate of false-positive results in pregnant women in Japan. PLoS ONE 5: e9382.

-Silverstein DM, et al. (2004) False-positive human immunodeficiency virus antibody test in a dialysis patient. Pediatr Nephrol 19: 547-549.

-Spencer DV, et al. (2009) Heterophilic antibody interference causing false-positive rapid human immunodeficiency virus antibody testing. Clin Chim Acta 399: 121-122.

-Wai CT, Tambyah PA (2002) False-positive HIV-1 ELISA in patients with hepatitis B. Am J Med 112: 737.

-Wesolowski LG, et al. (2011) False-positive human immunodeficiency virus enzyme immunoassay results in pregnant women. PLoS ONE 6: e16538.

-Willman JH, et al. (1999) Heterophile antibodies to bovine and caprine proteins causing false-positive human immunodeficiency virus type 1 and other enzyme-linked immunosorbent assay results. Clin Diagn Lab Immunol 6: 615-616.

-Zacharias NM, et al. (2004) High false-positive rate of human immunodeficiency virus rapid serum screening in a predominantly hispanic prenatal population. J Perinatol 24: 743-747.

-Zdeb MS (2007) HIV screening and false-positive results. Jama 297: 947-948; author reply 948.

-Zehender G, et al. (1997) High prevalence of false-negative anti-HTLV type I/II enzyme-linked immunosorbent assay results in HIV type 1-positive patients. AIDS Res Hum Retroviruses 13: 1141-1146.

%d bloggers like this: